Evidence mapPaperPMID 39225225Full record

ReviewCurrent protein & peptide science2025

Diet-induced Obesity: Pathophysiology, Consequences and Target Specific Therapeutic Strategies.

Munmun Banerjee, Veda P Pandey

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In one paragraph

Review in Current protein & peptide science, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Munmun BanerjeeDepartment of Biochemistry, University of Lucknow, Lucknow 226007, India.
Veda P PandeyDepartment of Biochemistry, University of Lucknow, Lucknow 226007, India.ORCID 0000-0002-5856-1519

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Diet has emerged as a pivotal factor in the current time for diet-induced obesity (DIO). A diet overloaded with fats and carbohydrates and unhealthy dietary habits contribute to the development of DIO through several mechanisms. The prominent ones include the transition of normal gut microbiota to obese microbiota, under-expression of AMPK, and abnormally high levels of adipogenesis. DIO is the root of many diseases. The present review deals with various aspects of DIO and its target proteins that can be specifically used for its treatment. Also, the currently available treatment strategies have been explored. It was found that the expression of five proteins, namely, PPARγ, FTO, CDK4, 14-3-3 ζ protein, and Galectin-1, is upregulated in DIO. They can be used as potential targets for drug-designing studies. Thus, with these targets, the treatment strategy for DIO using natural bioactive compounds can be a safer alternative to medications and bariatric surgeries.

Indexed as

DietObesityAdipogenesisAlpha-Ketoglutarate-Dependent Dioxygenase FTOAnimalsCyclin-Dependent Kinase 4Diet, High-FatGastrointestinal MicrobiomeHumansPPAR gammaAlpha-Ketoglutarate-Dependent Dioxygenase FTOCyclin-Dependent Kinase 4FTO protein, humanPPAR gamma14-3-3 ζ proteinCDK4Diet-induced obesityFTOGalectin-1.PPARγ

Identifiers

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.