Evidence mapPaperPMID 39225830Full record

ReviewNaunyn-Schmiedeberg's archives of pharmacology2025

Metformin and the PI3K/AKT signaling pathway: implications for cancer, cardiovascular, and central nervous system diseases.

Hemin Ashayeri Ahmadabad, Somayeh Mohammadi Panah, Hojat Ghasemnejad-Berenji, Shabnam Ghojavand, Morteza Ghasemnejad-Berenji, Mohammad Rafi Khezri

Abstract readReview
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In one paragraph

Review in Naunyn-Schmiedeberg's archives of pharmacology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Hemin Ashayeri AhmadabadSchool of Medicine, Islamic Azad University of Medical Science, Tehran, Iran.
Somayeh Mohammadi PanahSchool of Pharmacy, Urmia University of Medical Sciences, Urmia, Iran.
Hojat Ghasemnejad-BerenjiReproductive Health Research Center, Clinical Research Institute, Urmia University of Medical Sciences, Urmia, Iran.
Shabnam GhojavandFaculty of Pharmacy, Islamic Azad University of Tehran, Tehran, Iran.
Morteza Ghasemnejad-BerenjiDepartment of Pharmacology and Toxicology, School of Pharmacy, Urmia University of Medical Sciences, Urmia, Iran. Morteza.ghasemnejad@yahoo.com.ORCID 0000-0001-5672-9202
Mohammad Rafi KhezriReproductive Health Research Center, Clinical Research Institute, Urmia University of Medical Sciences, Urmia, Iran. drmnkh76@gmail.com.ORCID 0000-0002-4280-0378

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Recent findings have brought our understanding of diseases at the molecular level, highlighting upstream intracellular pathways as potential therapeutic targets. The PI3K/AKT pathway, a key regulator of cellular responses to environmental changes, is frequently altered in various diseases, making it a promising target for intervention. Metformin is the most known anti-diabetic agent that is known due to its effects on cancer, inflammatory-related diseases, oxidative stress, and other human diseases. It is clearly understood that metformin modulates the activity of the PI3K/AKT pathway leading to a wide variety of outcomes. This interaction has been well-studied in various diseases. Therefore, this review aims to examine PI3K/AKT-modulating properties of metformin in cancer, cardiovascular, and central nervous system diseases. Our findings indicate that metformin is effective in treating cancer and CNS diseases, and plays a role in both the prevention and treatment of cardiovascular diseases. These insights support the potential of metformin in comprehensive strategies for disease management.

Indexed as

Cardiovascular DiseasesCentral Nervous System DiseasesHypoglycemic AgentsMetforminNeoplasmsPhosphatidylinositol 3-KinasesProto-Oncogene Proteins c-aktAnimalsHumansSignal TransductionHypoglycemic AgentsMetforminPhosphatidylinositol 3-KinasesProto-Oncogene Proteins c-aktAKTCancerCardiovascular diseasesCNSMetforminPI3K

Identifiers

What Socratic holds

Texttitle and abstract
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.