Evidence mapPaperPMID 39249105Full record

ArticleJournal of neurology2024

Rapid differentiation of MOGAD and MS after a single optic neuritis.

T Pakeerathan, J Havla, C Schwake, A Salmen, M Ringelstein, O Aktas, M Weise, J A Gernert, B Kornek, G Bsteh and 16 more

Abstract readMulticenter Study
In one paragraph

Article in Journal of neurology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

26 authors.

T PakeerathanDepartment of Neurology, St. Josef-Hospital, Ruhr-University Bochum, Gudrunstr. 56, 44791, Bochum, Germany.
J HavlaInstitute of Clinical Neuroimmunology, LMU Hospital, Ludwig-Maximilians Universität München, Munich, Germany.
C SchwakeDepartment of Neurology, St. Josef-Hospital, Ruhr-University Bochum, Gudrunstr. 56, 44791, Bochum, Germany.
A SalmenDepartment of Neurology, St. Josef-Hospital, Ruhr-University Bochum, Gudrunstr. 56, 44791, Bochum, Germany.
M RingelsteinDepartment of Neurology, Medical Faculty, Heinrich-Heine-University Düsseldorf, Düsseldorf, Germany.
O AktasDepartment of Neurology, Medical Faculty, Heinrich-Heine-University Düsseldorf, Düsseldorf, Germany.
M WeiseDepartment of Neurology, Medical Faculty, Heinrich-Heine-University Düsseldorf, Düsseldorf, Germany.
J A GernertInstitute of Clinical Neuroimmunology, LMU Hospital, Ludwig-Maximilians Universität München, Munich, Germany.
B KornekDepartment of Neurology, Medical University of Vienna, Vienna, Austria.
G BstehDepartment of Neurology, Medical University of Vienna, Vienna, Austria.
A-K PröbstelDepartment of Neurology, University Hospital Basel and University of Basel, Basel, Switzerland.
A PapadopoulouDepartment of Neurology, University Hospital Basel and University of Basel, Basel, Switzerland.
L KulsvehagenDepartment of Neurology, University Hospital Basel and University of Basel, Basel, Switzerland.
A B Ayroza Galvão Ribeiro GomesDepartment of Neurology, University Hospital Basel and University of Basel, Basel, Switzerland.
N Cerdá-FuertesDepartment of Biomedicine and Clinical Research, University Hospital Basel and University of Basel, Basel, Switzerland.
F C OertelNeuroscience Clinical Research Center, Charité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany.
A S DuchowNeuroscience Clinical Research Center, Charité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany.
F PaulNeuroscience Clinical Research Center, Charité-Universitätsmedizin Berlin, corporate member of Freie Universität Berlin and Humboldt-Universität zu Berlin, Berlin, Germany.
J P StellmannAPHM, Hopital de La Timone, CEMEREM, Marseille, France.
N StolowyDepartment of Ophthalmology, Centre Hospitalier Universitaire de La Timone, Marseille, France.
K HellwigDepartment of Neurology, St. Josef-Hospital, Ruhr-University Bochum, Gudrunstr. 56, 44791, Bochum, Germany.
C Schneider-GoldDepartment of Neurology, St. Josef-Hospital, Ruhr-University Bochum, Gudrunstr. 56, 44791, Bochum, Germany.
T KümpfelInstitute of Clinical Neuroimmunology, LMU Hospital, Ludwig-Maximilians Universität München, Munich, Germany.
R GoldDepartment of Neurology, St. Josef-Hospital, Ruhr-University Bochum, Gudrunstr. 56, 44791, Bochum, Germany.
P AlbrechtDepartment of Neurology, Medical Faculty, Heinrich-Heine-University Düsseldorf, Düsseldorf, Germany.
I AyzenbergDepartment of Neurology, St. Josef-Hospital, Ruhr-University Bochum, Gudrunstr. 56, 44791, Bochum, Germany. ilya.ayzenberg@rub.de.ORCID http://orcid.org/0000-0002-6009-792X

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundOptic neuritis (ON) is a common manifestation of multiple sclerosis (MS) and myelin-oligodendrocyte-glycoprotein IgG-associated disease (MOGAD). This study evaluated the applicability of optical coherence tomography (OCT) for differentiating between both diseases in two independent cohorts.

methodsOne hundred sixty two patients from seven sites underwent standard OCT and high-contrast visual acuity (HCVA) testing at least 6 months after first ON. Of these, 100 patients (32 MOGAD, 68 MS) comprised the primary investigational cohort, while 62 patients (31 MOGAD, 31 MS) formed a validation cohort. A composite score distinguishing between MOGAD and MS was developed using multivariate logistic regression.

resultsBilateral simultaneous ON occurred more frequently in MOGAD compared to MS (46.9 vs. 11.8%, p < 0.001). OCT revealed more peripapillary retinal nerve fiber layer (pRNFL) atrophy in all segments in MOGAD compared to predominantly temporal pRNFL atrophy in MS (p < 0.001). HCVA was better preserved in MS (p = 0.007). pRNFL thickness in all except for temporal segments was suitable for differentiating MOGAD and MS. Simultaneous bilateral ON and critical atrophy in nasal (< 58.5 µm) and temporal superior (< 105.5 µm) segments were included into the composite score as three independent predictors for MOGAD. The composite score distinguished MOGAD from MS with 75% sensitivity and 90% specificity in the investigational cohort, and 68% sensitivity and 87% specificity in the validation cohort.

conclusionFollowing a single ON-episode, MOGAD exhibits more pronounced global pRNFL atrophy and lower visual acuity after ON compared to MS. The introduced OCT-based composite score enabled differentiation between the two entities across both cohorts.

Indexed as

Multiple SclerosisMyelin-Oligodendrocyte GlycoproteinOptic NeuritisTomography, Optical CoherenceAdultCohort StudiesDemyelinating Autoimmune Diseases, CNSDiagnosis, DifferentialFemaleHumansMaleMiddle AgedVisual AcuityMyelin-Oligodendrocyte GlycoproteinMultiple sclerosisMyelin-oligodendrocyte-glycoprotein IgGMyelin oligodendrocyte glycoprotein IgG-associated diseaseOptical coherence tomographyOptic neuritisVisual evoked potential

Identifiers

PMID39249105
PMCPMC11561115

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.