Evidence map›Paper›PMID 39249107›Full record

ArticleJournal of neurology2024

Plasma NfL, GFAP, amyloid, and p-tau species as Prognostic biomarkers in Parkinson's disease.

Andrea Pilotto, Nicholas J Ashton, Alessandro Lupini, Beatrice Battaglio, Cinzia Zatti, Chiara Trasciatti, Stefano Gipponi, Elisabetta Cottini, Ilaria Grossi, Alessandro Salvi and 6 more

Abstract read
In one paragraph

Article in Journal of neurology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 27 papers.

0numbers the graph read from it
0cells of the map it votes in
27citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

27 citing papers in PubMed.

  1. Article
  2. A multiverse analysis of the logical memory test and plasma biomarkers of Alzheimer's disease.Cortex; a journal devoted to the study of the nervous system and behavior · 2026
    Article
  3. What's in a name? reframing advanced Parkinson's disease as a multidimensional state.Journal of neural transmission (Vienna, Austria : 1996) · 2026
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors.

Andrea Pilotto *Neurology Unit, Department of Clinical and Experimental Sciences, University of Brescia, P.Zzale Spedali Civili, 1, 25123, Brescia, Italy. Andrea.pilotto@unibs.it.ORCID http://orcid.org/0000-0003-2029-6606
Nicholas J Ashton *Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
Alessandro Lupini *Neurology Unit, Department of Clinical and Experimental Sciences, University of Brescia, P.Zzale Spedali Civili, 1, 25123, Brescia, Italy.
Beatrice BattaglioNeurology Unit, Department of Clinical and Experimental Sciences, University of Brescia, P.Zzale Spedali Civili, 1, 25123, Brescia, Italy.
Cinzia ZattiNeurology Unit, Department of Clinical and Experimental Sciences, University of Brescia, P.Zzale Spedali Civili, 1, 25123, Brescia, Italy.
Chiara TrasciattiNeurology Unit, Department of Clinical and Experimental Sciences, University of Brescia, P.Zzale Spedali Civili, 1, 25123, Brescia, Italy.
Stefano GipponiNeurology Unit, Department of Clinical and Experimental Sciences, University of Brescia, P.Zzale Spedali Civili, 1, 25123, Brescia, Italy.
Elisabetta CottiniDepartment of Continuity of Care and Frailty, Neurology Unit, ASST Spedali Civili Hospital, Brescia, Italy.
Ilaria GrossiDivision of Biology and Genetics, Department of Molecular and Translational Medicine, University of Brescia, Brescia, Italy.
Alessandro SalviDivision of Biology and Genetics, Department of Molecular and Translational Medicine, University of Brescia, Brescia, Italy.
Giuseppina de PetroDivision of Biology and Genetics, Department of Molecular and Translational Medicine, University of Brescia, Brescia, Italy.
Marina PizziDivision of Pharmacology, Department of Molecular and Translational Medicine, University of Brescia, Brescia, Italy.
Antonio CanaleDepartment of Statistical Sciences, University of Padova, Padua, Italy.
Kaj BlennowDepartment of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
Henrik ZetterbergDepartment of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, The Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.
Alessandro PadovaniNeurology Unit, Department of Clinical and Experimental Sciences, University of Brescia, P.Zzale Spedali Civili, 1, 25123, Brescia, Italy.

Funding

Research Education ComponentP30AG072980 · NIA · BANNER HEALTH · PI ALIREZA ATRI · 2021 to 2026
$24.9M
The effects of iron on oxidative stress and Alzheimer's biomarkers in amyloid-positive and negative elderly normalR01AG068398 · NIA · WEILL MEDICAL COLL OF CORNELL UNIV · PI CHIANG, GLORIA CHIA-YI · 2020 to 2024
$2.8M
AD Strategic Fund and the Alzheimer's Association ADSF-21-831376-CAD Strategic Fund and the Alzheimer's Association ADSF-21-831377-CAD Strategic Fund and the Alzheimer's Association ADSF-21-831381-CAiralzh Foundation AGYR2021ALF-agreement ALFGBG-715986Alzheimer Drug Discovery Foundation ADDFEU Joint Programme - Neurodegenerative Disease Research JPND2021-00694European Union Joint Program for Neurodegenerative Disorders JPND2019-466-236H2020 European Research Council ID853981H2020 Marie Skłodowska-Curie Actions 860197 (MIRIADE)Health and Wealth research grants 2016 of the University of Brescia DMAHealth and Wealth research grants 2016 of the University of Brescia number NP 1471Hjärnfonden FO2017-0243HORIZON EUROPE Reforming and enhancing the European Research and Innovation system 101053962Ministero della Salute RF-2018-12366209Ministero dell'Istruzione, dell'Università e della Ricerca 2017MYJ5THNIA NIH HHS P30 AG072980NIA NIH HHS R01 AG068398Office of Dietary Supplements 1R01AG068398-01PNRR-Health PNRR-MAD-2022-12376110RePlast PRIN 2021Stiftelsen för Gamla Tjänarinnor FO2022-0270Swedish Alzheimer Foundation AF-742881Swedish Research Council 2017-00915Swedish Research Council 2019-02397Swedish Research Council 2022-01018Swedish State Support for Clinical Research ALFGBG-71320UCLH Biomedical Research Centre UKDRI-1003
6 · The paper itself

Abstract

introductionThe prognostic role of plasma neurofilament light chain (NfL), phospho-tau, beta-amyloid, and GFAP is still debated in Parkinson's disease (PD).

methodsPlasma p-tau181, p-tau231, Aβ1-40, Aβ1-42, GFAP, and NfL were measured by SIMOA in 136 PD with 2.9 + 1.7 years of follow-up and 76 controls. Differences in plasma levels between controls and PD and their correlation with clinical severity and progression rates were evaluated using linear regression analyses.

resultsPatients exhibited similar distribution of plasma biomarkers but higher P-tau181, P-tau231 and lower Aβ1-42 compared with controls. NfL and GFAP correlated with baseline motor and non-motor severity measures. At follow-up, NfL emerged as the best predictor of progression with marginal effect of GFAP and p-tau181 adjusting for age, sex, disease duration, and baseline motor severity.

conclusionThe present findings confirmed plasma NfL as best predictor of progression in PD, with a marginal role of p-tau181 and GFAP.

Indexed as

Amyloid beta-PeptidesBiomarkersDisease ProgressionGlial Fibrillary Acidic ProteinNeurofilament ProteinsParkinson Diseasetau ProteinsAgedFemaleFollow-Up StudiesHumansMaleMiddle AgedPeptide FragmentsPhosphorylationPrognosisAmyloid beta-Peptidesamyloid beta-protein (1-42)BiomarkersGFAP protein, humanGlial Fibrillary Acidic ProteinMAPT protein, humanneurofilament protein LNeurofilament ProteinsPeptide Fragmentstau ProteinsGFAPNeurofilament light chainParkinson’s diseasePhosphorylated tauPlasma biomarkersProgression

Identifiers

PMID39249107
PMCPMC11588809

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.