Evidence map›Paper›PMID 39264521›Full record

ArticleCardiovascular toxicology2024

The Worsening of Myocardial Ischemia-Reperfusion Injury in Uremic Cardiomyopathy is Further Aggravated by PM

Bhavana Sivakumar, Gino A Kurian

Abstract read
PubMed Publisher
In one paragraph

Article in Cardiovascular toxicology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Bhavana SivakumarVascular Biology Lab, School of Chemical and Biotechnology, SASTRA Deemed University, Thanjavur, Tamil Nadu, India.
Gino A KurianVascular Biology Lab, School of Chemical and Biotechnology, SASTRA Deemed University, Thanjavur, Tamil Nadu, India. ginokurian@hotmail.com.

Funding

Department of Science and Technology, Ministry of Science and Technology, India CRG/2021/000227Indian council of medical research 3/1/2(22)/Env/2021-NCD-II
6 · The paper itself

Abstract

Uremic cardiomyopathy (UC) represents a complex syndrome characterized by different cardiac complications, including systolic and diastolic dysfunction, left ventricular hypertrophy, and diffuse fibrosis, potentially culminating in myocardial infarction (MI). Revascularization procedures are often necessary for MI management and can induce ischemia reperfusion injury (IR). Despite this clinical relevance, the role of fine particulate matter (PM

Indexed as

ApoptosisCardiomyopathiesDisease Models, AnimalEnergy MetabolismMitochondria, HeartMyocardial Reperfusion InjuryParticulate MatterSignal TransductionUremiaAdenineAir PollutantsAnimalsMaleMyocardial InfarctionMyocytes, CardiacOxidative StressAdenineAir PollutantsParticulate MatterPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaPhosphatidylinositol 3-KinasePpargc1a protein, ratProto-Oncogene Proteins c-aktMitochondriaMyocardial ischemia reperfusion injuryOxidative stressPM2.5Uremic cardiomyopathy

Identifiers

PMID39264521

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.