Evidence map›Paper›PMID 39299707›Full record

ArticleJournal of child psychology and psychiatry, and allied disciplines2025

Nature and nurture in fussy eating from toddlerhood to early adolescence: findings from the Gemini twin cohort.

Zeynep Nas, Moritz Herle, Alice R Kininmonth, Andrea D Smith, Rachel Bryant-Waugh, Alison Fildes, Clare H Llewellyn

Abstract readTwin Study
In one paragraph

Article in Journal of child psychology and psychiatry, and allied disciplines, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
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  12. Helping parents to cope with infant regulatory disorders.Frontiers in child and adolescent psychiatry · 2024
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Zeynep NasDepartment of Behavioural Science & Health, University College London, London, UK.ORCID 0000-0002-6675-621X
Moritz HerleSocial, Genetic & Developmental Psychiatry Centre, Institute of Psychiatry, Psychology, and Neuroscience, King's College London, London, UK.
Alice R KininmonthDepartment of Behavioural Science & Health, University College London, London, UK.
Andrea D SmithMRC Epidemiology Unit, University of Cambridge, Cambridge, UK.
Rachel Bryant-WaughSouth London and Maudsley NHS Foundation Trust, London, UK.ORCID 0000-0002-7449-6059
Alison FildesSchool of Psychology, University of Leeds, Leeds, UK.
Clare H LlewellynDepartment of Behavioural Science & Health, University College London, London, UK.

Funding

MQ Mental Health Research MPSIP/6MQ Mental Health Research MQF17/4Rosetrees Trust M749
6 · The paper itself

Abstract

backgroundFood fussiness (FF) describes the tendency to eat a small range of foods, due to pickiness and/or reluctance to try new foods. A common behaviour during childhood, and a considerable cause of caregiver concern; its causes are poorly understood. This is the first twin study of genetic and environmental contributions to the developmental trajectory of FF from toddlerhood to early adolescence, and stability and change over time.

methodsParticipants were from Gemini, a population-based British cohort of n = 4,804 twins born in 2007. Parents reported on FF using the Child Eating Behaviour Questionnaire 'FF' scale when children were 16 months (n = 3,854), 3 (n = 2,666), 5 (n = 2,098), 7 (n = 703), and 13 years old (n = 970). A mixed linear model examined the trajectory of FF, and a correlated factors twin model quantified genetic and environmental contributions to variation in and covariation between trajectory parameters. A longitudinal Cholesky twin model examined genetic and environmental influences on FF at each discrete age.

resultsWe modelled a single FF trajectory for all children, which was characterised by increases from 16 months to 7 years, followed by a slight decline from 7 to 13 years. All trajectory parameters were under strong genetic influence (>70%) that was largely shared, indicated by high genetic correlations. Discrete age analyses showed that genetic influence on FF increased significantly after toddlerhood (16 months: 60%, 95% CI: 53%-67%; 3 years: 83%; 81%-86%), with continuing genetic influence as indicated by significant genetic overlap across every age. Shared environmental influences were only significant during toddlerhood. Unique environmental influences explained 15%-26% of the variance over time, with some enduring influence from 5 years onwards.

conclusionsIndividual differences in FF were largely explained by genetic factors at all ages. Fussy eating also shows a significant proportion of environmental influence, especially in toddlerhood, and may, therefore, benefit from early interventions throughout childhood. Future work needs to refine the FF trajectory and explore specific trajectory classes.

Indexed as

Child BehaviorChild DevelopmentFood FussinessGene-Environment InteractionAdolescentChildChild, PreschoolFemaleHumansInfantLongitudinal StudiesMaleUnited Kingdomeating behaviourLongitudinal studiestwins

Identifiers

PMID39299707
PMCPMC11754699

What Socratic holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.