Evidence map›Paper›PMID 39301664›Full record

SynthesisDiabetes & metabolism journal2025

Identification and Potential Clinical Utility of Common Genetic Variants in Gestational Diabetes among Chinese Pregnant Women.

Claudia Ha-Ting Tam, Ying Wang, Chi Chiu Wang, Lai Yuk Yuen, Cadmon King-Poo Lim, Junhong Leng, Ling Wu, Alex Chi-Wai Ng, Yong Hou, Kit Ying Tsoi and 9 more

Abstract readMeta-Analysis
In one paragraph

Synthesis in Diabetes & metabolism journal, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

  1. Article
  2. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

19 authors.

Claudia Ha-Ting TamDepartment of Medicine and Therapeutics, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Ying WangScientific Research Platform of the Second School of Clinical Medicine, Guangdong Medical University, Dongguan, China.
Chi Chiu WangDepartment of Obstetrics and Gynecology, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Lai Yuk YuenDepartment of Obstetrics and Gynecology, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Cadmon King-Poo LimDepartment of Medicine and Therapeutics, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Junhong LengDepartment of Children's Health, Tianjin Women and Children's Health Center, Tianjin, China.
Ling WuDepartment of Obstetrics and Gynecology, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Alex Chi-Wai NgDepartment of Medicine and Therapeutics, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Yong HouDepartment of Medicine and Therapeutics, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Kit Ying TsoiDepartment of Medicine and Therapeutics, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Hui WangDepartment of Epidemiology and Biostatistics, School of Public Health, Tianjin Medical University, Tianjin, China.
Risa OzakiDepartment of Medicine and Therapeutics, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Albert Martin LiDepartment of Pediatrics, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Qingqing WangDepartment of Obstetrics and Gynecology, The Third Affiliated Hospital of Sun Yat-Sen University, Guangzhou, China.
Juliana Chung-Ngor ChanDepartment of Medicine and Therapeutics, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Yan Chou YeDepartment of Obstetrics and Gynecology, The Seventh Affiliated Hospital of Sun Yat-Sen University, Shenzhen, China.
Wing Hung TamDepartment of Obstetrics and Gynecology, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.
Xilin YangDepartment of Epidemiology and Biostatistics, School of Public Health, Tianjin Medical University, Tianjin, China.
Ronald Ching-Wan MaDepartment of Medicine and Therapeutics, Prince of Wales Hospital, The Chinese University of Hong Kong, Hong Kong, China.

Funding

Leveraging innovative technologies in basic and clinical cancer researchT32CA009302 · NCI · STANFORD UNIVERSITY · PI LAURA D ATTARDI, Aaron M Newman · 1985 to 2026
$19.1M
Hyperglycemia and Adverse Pregnancy Outcome (HAPO)R01HD034243 · NICHD · NORTHWESTERN UNIVERSITY AT CHICAGO · PI METZGER, BOYD E · 1999 to 2010
$16.2M
HAPO: DATA COORDINATING CENTERR01HD034242 · NICHD · NORTHWESTERN UNIVERSITY AT CHICAGO · PI DYER, ALAN R · 1999 to 2007
$3.0M
Council of the Hong Kong SAR 14102719Council of the Hong Kong SAR 14118718Council of the Hong Kong SAR 471713Council of the Hong Kong SAR 473408National Institute of Child Health and Human Development R01-HD34242NCI NIH HHS T32 CA009302NICHD NIH HHS R01 HD034242NICHD NIH HHS R01 HD034243NIDDK NIH HHS R01-HD34243Research Impact Fund R4012-18RGC Theme-based Research Scheme T12-402/13 NUniversity Grants Committee Research Grants Matching Scheme
6 · The paper itself

Abstract

backgruoundThe genetic basis for hyperglycaemia in pregnancy remain unclear. This study aimed to uncover the genetic determinants of gestational diabetes mellitus (GDM) and investigate their applications.

methodsWe performed a meta-analysis of genome-wide association studies (GWAS) for GDM in Chinese women (464 cases and 1,217 controls), followed by de novo replications in an independent Chinese cohort (564 cases and 572 controls) and in silico replication in European (12,332 cases and 131,109 controls) and multi-ethnic populations (5,485 cases and 347,856 controls). A polygenic risk score (PRS) was derived based on the identified variants.

resultsUsing the genome-wide scan and candidate gene approaches, we identified four susceptibility loci for GDM. These included three previously reported loci for GDM and type 2 diabetes mellitus (T2DM) at MTNR1B (rs7945617, odds ratio [OR], 1.64; 95% confidence interval [CI], 1.38 to 1.96), CDKAL1 (rs7754840, OR, 1.33; 95% CI, 1.13 to 1.58), and INS-IGF2-KCNQ1 (rs2237897, OR, 1.48; 95% CI, 1.23 to 1.79), as well as a novel genome-wide significant locus near TBR1-SLC4A10 (rs117781972, OR, 2.05; 95% CI, 1.61 to 2.62; Pmeta=7.6×10-9), which has not been previously reported in GWAS for T2DM or glycaemic traits. Moreover, we found that women with a high PRS (top quintile) had over threefold (95% CI, 2.30 to 4.09; Pmeta=3.1×10-14) and 71% (95% CI, 1.08 to 2.71; P=0.0220) higher risk for GDM and abnormal glucose tolerance post-pregnancy, respectively, compared to other individuals.

conclusionOur results indicate that the genetic architecture of glucose metabolism exhibits both similarities and differences between the pregnant and non-pregnant states. Integrating genetic information can facilitate identification of pregnant women at a higher risk of developing GDM or later diabetes.

Indexed as

Diabetes, GestationalAdultCase-Control StudiesChinaDiabetes Mellitus, Type 2East Asian PeopleFemaleGenetic Predisposition to DiseaseGenetic VariationGenome-Wide Association StudyHumansPolymorphism, Single NucleotidePregnancyReceptor, Melatonin, MT2tRNA MethyltransferasesCDKAL1 protein, humanMTNR1B protein, humanReceptor, Melatonin, MT2tRNA MethyltransferasesDiabetes, gestationalGenetic risk scoreGenome-wide association studyGlucose intolerancePregnant women

Identifiers

PMID39301664
PMCPMC11788552

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.