Evidence map›Paper›PMID 39309892›Full record

ArticleHeliyon2024

Enhancing terminal erythroid differentiation in human embryonic stem cells through TRIB3 overexpression.

Xiaoling Wang, Tiantian Cui, Hao Yan, Lingping Zhao, Ruge Zang, Hongyu Li, Haiyang Wang, Biao Zhang, Junnian Zhou, Yiming Liu and 3 more

Abstract read
In one paragraph

Article in Heliyon, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Xiaoling WangBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Tiantian CuiBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Hao YanBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Lingping ZhaoBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Ruge ZangBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Hongyu LiBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Haiyang WangBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Biao ZhangBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Junnian ZhouBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Yiming LiuBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Wen YueBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Jiafei XiBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.
Xuetao PeiBeijing Institute of Radiation Medicine, Beijing, 100850, PR China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Tribbles pseudokinase 3 (TRIB3) expression significantly increases during terminal erythropoiesis in vivo. However, we found that TRIB3 expression remained relatively low during human embryonic stem cell (hESC) erythropoiesis, particularly in the late stage, where it is typically active. TRIB3 was expressed in megakaryocyte-erythrocyte progenitor cells and its low expression was necessary for megakaryocyte differentiation. Thus, we proposed that the high expression during late stage of erythropoiesis could be the clue for promotion of maturation of hESC-derived erythroid cells. To our knowledge, the role of TRIB3 in the late stage of erythropoiesis remains ambiguous. To address this, we generated inducible TRIB3 overexpression hESCs, named TRIB3

Indexed as

Human embryonic stem cellMitophagyTerminal erythropoiesisTribbles pseudokinase 3

Identifiers

PMID39309892
PMCPMC11415673

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.