Evidence mapPaperPMID 39337461Full record

ReviewInternational journal of molecular sciences2024

New Insights into Mitochondria in Health and Diseases.

Ya Li, Huhu Zhang, Chunjuan Yu, Xiaolei Dong, Fanghao Yang, Mengjun Wang, Ziyuan Wen, Mohan Su, Bing Li, Lina Yang

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed.

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  14. Hemistepsin A induces apoptosis by modulating the reactive oxygen species-dependent PI3K/Akt signaling pathway in human lung carcinoma A549 cells.The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology · 2025
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Ya LiDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.
Huhu ZhangDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.
Chunjuan YuDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.
Xiaolei DongDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.
Fanghao YangDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.
Mengjun WangDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.
Ziyuan WenDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.
Mohan SuDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.
Bing LiDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.
Lina YangDepartment of Genetics and Cell Biology, Basic Medical College, Qingdao University, Qingdao 266071, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Mitochondria are a unique type of semi-autonomous organelle within the cell that carry out essential functions crucial for the cell's survival and well-being. They are the location where eukaryotic cells carry out energy metabolism. Aside from producing the majority of ATP through oxidative phosphorylation, which provides essential energy for cellular functions, mitochondria also participate in other metabolic processes within the cell, such as the electron transport chain, citric acid cycle, and β-oxidation of fatty acids. Furthermore, mitochondria regulate the production and elimination of ROS, the synthesis of nucleotides and amino acids, the balance of calcium ions, and the process of cell death. Therefore, it is widely accepted that mitochondrial dysfunction is a factor that causes or contributes to the development and advancement of various diseases. These include common systemic diseases, such as aging, diabetes, Parkinson's disease, and cancer, as well as rare metabolic disorders, like Kearns-Sayre syndrome, Leigh disease, and mitochondrial myopathy. This overview outlines the various mechanisms by which mitochondria are involved in numerous illnesses and cellular physiological activities. Additionally, it provides new discoveries regarding the involvement of mitochondria in both disorders and the maintenance of good health.

Indexed as

MitochondriaMitochondrial DiseasesAnimalsEnergy MetabolismHumansNeoplasmsReactive Oxygen SpeciesReactive Oxygen Speciesagingbioenergeticsmitochondrialmitochondrial dysfunctionmitochondrial targeted therapymtDNAmutationsROS

Identifiers

PMID39337461
PMCPMC11432609

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.