Evidence mapPaperPMID 39347908Full record

ArticleJournal of endocrinological investigation2025

Interleukin-5: an indicator of mild cognitive impairment in patients with type 2 diabetes mellitus - a comprehensive investigation ranging from bioinformatics analysis to clinical research.

Hui Zhang, Wenwen Zhu, Shufang Yang, Tong Niu, Huzaifa Fareeduddin Mohammed Farooqui, Bing Song, Hongxiao Wang, Sumei Li, Jumei Wang, Linlin Xu and 2 more

Abstract read
In one paragraph

Article in Journal of endocrinological investigation, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Article
  2. Article
  3. Role of Interleukins in Type 1 and Type 2 Diabetes.Diagnostics (Basel, Switzerland) · 2025
    Review
  4. Article
  5. Article
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Hui Zhang *Henan Key Laboratory of Rare Diseases, Endocrinology and Metabolism Center, The First Affiliated Hospital, College of Clinical Medicine of Henan, University of Science and Technology, Luoyang, China.
Wenwen Zhu *Department of Endocrinology, Affiliated Zhongda Hospital of Southeast University, Nanjing, China.
Shufang Yang *Department of Endocrinology, Taizhou People's Hospital, Taizhou, China.
Tong NiuDepartment of Endocrinology, Affiliated Zhongda Hospital of Southeast University, Nanjing, China.
Huzaifa Fareeduddin Mohammed FarooquiDepartment of Endocrinology, Affiliated Zhongda Hospital of Southeast University, Nanjing, China.
Bing SongDepartment of Endocrinology, The First Affiliated Hospital of Jinzhou Medical University, Jinzhou, China.
Hongxiao WangDepartment of Endocrinology, The First Affiliated Hospital of Jinzhou Medical University, Jinzhou, China.
Sumei LiDepartment of Endocrinology, Institute of Endocrine and Metabolic Diseases, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, China.
Jumei WangDepartment of Endocrinology, Institute of Endocrine and Metabolic Diseases, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, China.
Linlin XuDepartment of Endocrinology, Institute of Endocrine and Metabolic Diseases, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, China.
Zhen ZhangDepartment of Endocrinology, Institute of Endocrine and Metabolic Diseases, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, China.
Haoqiang ZhangDepartment of Endocrinology, Institute of Endocrine and Metabolic Diseases, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, 230001, China. drhqzhang@ustc.edu.cn.ORCID http://orcid.org/0000-0001-6937-4252

Funding

China Endocrine Metabolism Talent Research Project from China International Medical Foundation 2023-N-03-12Research Funds of Center for Leading Medicine and Advanced Technologies of IHM 2023IHM02006Scientific Research Start-up Funds of The First Affiliated Hospital of USTC RC2021178
6 · The paper itself

Abstract

purposeNeuroinflammation constitutes an underlying mechanism for cognitive impairment. Here, we endeavor to scrutinize the potential contribution of interleukin-5 (IL-5) towards mild cognitive impairment (MCI), and to assess its diagnostic value for MCI in patients with type 2 diabetes mellitus (T2DM).

methodsRNA-seq was used to explore the potential neuroinflammation factors in the hippocampus of diabetic mice with cognitive decline. Additionally, the promising risk factor was verified in animals. Finally, the association between IL-5 levels and cognitive function and its diagnostic value for MCI were assessed.

resultsIn animals, up-regulated IL-5 mRNA and protein levels were detected by RNA-seq and (or) verified experiments in the hippocampus of diabetic db/db mice with cognitive decline, compared to those of db/m mice without diabetes. In human, compared to diabetic patients without MCI, those with MCI demonstrate elevated levels of IL-5. It is natively associated with Montreal Cognitive Assessment (MoCA) scores, reflecting global cognitive function, and positively correlated with Trail Making Test A (TMTA) scores, reflecting information processing speed. Furthermore, an elevated level of IL-5 is identified as a risk factor for MCI, and a factor that influences TMTA scores. Finally, it is recommended that the cut-off value for IL-5 in the diagnosis of MCI is 22.98 pg/mL, with a sensitivity of 68.6% and specificity of 72.9%.

conclusionsIL-5 is considered a risk factor for MCI in T2DM patients and is associated with their performance in information processing speed. Moreover, an elevated level of IL-5 is a plausible biomarker for MCI in T2DM patients.

Indexed as

Cognitive DysfunctionDiabetes Mellitus, Type 2Interleukin-5AgedAnimalsBiomarkersComputational BiologyFemaleHippocampusHumansMaleMiceMiddle AgedBiomarkersIL5 protein, humanInterleukin-5Interleukin-5Mild cognitive impairmentNeuroinflammationType 2 diabetes mellitus

Identifiers

PMID39347908
PMCPMC11785607

What Socratic holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.