Evidence mapPaperPMID 39372196Full record

ArticleFrontiers in pharmacology2024

Protective effect of MP-40 mitigates BDL-induced hepatic fibrosis by inhibiting the NLRP3-mediated pyroptosis.

Xuedong Wan, Yuanyuan Fang, Minjing Qin, Qitong Zheng, Qiao Yang, Mengyun Peng, Min Hao, Kuilong Wang, Ruihua Zhao, Yiqing Shi and 3 more

Abstract read
In one paragraph

Article in Frontiers in pharmacology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Review
  2. Review
  3. Periodontitis and MASLD: a narrative review of the direct oral-hepatic pathway.Frontiers in cellular and infection microbiology · 2026
    Review
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Xuedong Wan *School of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Yuanyuan Fang *School of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Minjing Qin *School of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Qitong ZhengSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Qiao YangSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Mengyun PengSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Min HaoSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Kuilong WangSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Ruihua ZhaoSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Yiqing ShiSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Xin HanSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Xia'nan SangSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.
Gang CaoSchool of Pharmacy, Zhejiang Chinese Medical University, Hangzhou, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: Hepatic fibrosis and its associated consequences continue to pose a substantial global health challenge. Developing novel approaches to hepatic fibrosis management and prevention is critically necessary. Radix Paeoniae Alba (RPA) is widely used in Traditional Chinese Medicine (TCM) to treat various diseases. Our earlier research found that a bioactive component of RPA had a dose-dependent effect on anti-allergic asthma. RPA reduces allergic asthma by slowing the hepatic wind, according to "Treatise on Febrile Diseases". However, this bioactive fraction's pharmacological effects and mechanisms on the liver are unknown. Aim: This study examined the bioactive fraction MP-40, the methanol extract of RPA (MRPA), on bile duct ligation (BDL) for its anti-hepatic fibrosis activity and potential mechanisms. Methods: First, the effectiveness of MP-40 in treating BDL-induced hepatic fibrosis in mice and rats was evaluated through survival rates, ALT, AST HYP, and pathological changes. Molecular assays were performed using Results: The findings demonstrated that MP-40 and MRPA could lower ALT, AST, and HYP levels, boost survival rates, and reduce liver damage in BDL mice and rats. Furthermore, MP-40 outperforms MRPA. MP-40 was proven to drastically diminish fibrotic α-SMA and Collagen I. The expression of pyroptosis-related proteins NLRP3, Cleaved Caspase-1, TGF-β1, GSDMD-N, and 1L-1β decreased. MP-40 inhibited the synthesis of pyroptosis-related proteins more effectively than MCC950 (an NLRP3-specific inhibitor). Monoterpene glycosides and tannins were shown to be the most potent MP-40 components. Finally, the delegate compounds MP-40, PF, and TGG were shown to have substantial inhibitory effects on HSC-T6 activation. Conclusion: The results proved that MP-40 alleviates BDL-induced cholestatic hepatic fibrosis by inhibiting NLRP3-mediated pyroptosis. PF and TGG play a role in treating BDL-induced cholestatic hepatic fibrosis in MP-40.

Indexed as

bile duct ligationhepatic fibrosisMP-40NLRP3Radix Paeoniae Alba

Identifiers

PMID39372196
PMCPMC11449770

What Socratic holds

Textmetadata
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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.