Article in Life science alliance, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
13 authors.
Melissa DamieckiInstitute of Biochemistry and Molecular Biology I, Medical Faculty and University Hospital Düsseldorf, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0002-3935-6552
Ritam NahaInstitute of Biochemistry and Molecular Biology I, Medical Faculty and University Hospital Düsseldorf, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0001-8822-409X
Yulia SchaumkesselInstitute of Biochemistry and Molecular Biology I, Medical Faculty and University Hospital Düsseldorf, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0009-0000-6016-3301
Philipp WesthoffInstitute of Plant Biochemistry, Cluster of Excellence on Plant Sciences (CEPLAS), Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0002-3494-9420
Nika AtanelovInstitute of Biochemistry and Molecular Biology I, Medical Faculty and University Hospital Düsseldorf, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.
Anja StefanskiMolecular Proteomics Laboratory, Medical Faculty and University Hospital, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0001-8532-954X
Patrick PetzschGenomics and Transcriptomics Laboratory, BMFZ, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0002-8355-5524
Kai StühlerMolecular Proteomics Laboratory, Medical Faculty and University Hospital, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.
Karl KöhrerGenomics and Transcriptomics Laboratory, BMFZ, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.
Andreas Pm WeberInstitute of Plant Biochemistry, Cluster of Excellence on Plant Sciences (CEPLAS), Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0003-0970-4672
Ruchika AnandInstitute of Biochemistry and Molecular Biology I, Medical Faculty and University Hospital Düsseldorf, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0001-7337-6007
Andreas S ReichertInstitute of Biochemistry and Molecular Biology I, Medical Faculty and University Hospital Düsseldorf, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.ORCID 0000-0001-9340-3113
Arun Kumar KondadiInstitute of Biochemistry and Molecular Biology I, Medical Faculty and University Hospital Düsseldorf, Heinrich Heine University Düsseldorf, Düsseldorf, Germany kondadi@hhu.de.ORCID 0000-0002-5888-7834
Funding
No grant is acknowledged in the PubMed record.
6 · The paper itself
Abstract
Mitochondria play central roles in metabolism and metabolic disorders such as type 2 diabetes. MIC26, a mitochondrial contact site and cristae organising system complex subunit, was linked to diabetes and modulation of lipid metabolism. Yet, the functional role of MIC26 in regulating metabolism under hyperglycemia is not understood. We used a multi-omics approach combined with functional assays using WT and
Indexed as
GlycolysisLipid MetabolismMitochondriaOxidative PhosphorylationAnimalsCholesterolDiabetes Mellitus, Type 2Energy MetabolismGlucoseHumansHyperglycemiaMiceMitochondrial ProteinsCholesterolGlucoseMitochondrial Proteins
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.
Mitochondrial apolipoprotein MIC26 is a metabolic rheostat regulating central cellular fuel pathways. · full record | Socratic