Evidence mapPaperPMID 39433211Full record

ReviewProgress in retinal and eye research2025

Eye on the horizon: The metabolic landscape of the RPE in aging and disease.

David S Hansman, Jianhai Du, Robert J Casson, Daniel J Peet

Abstract readReview
In one paragraph

Review in Progress in retinal and eye research, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers.

0numbers the graph read from it
0cells of the map it votes in
23citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

23 citing papers in PubMed.

  1. Review
  2. Review
  3. Article
  4. Polyunsaturated fatty acid metabolism in the retinal pigment epithelium and its association with outer retinal disease.Mammalian genome : official journal of the International Mammalian Genome Society · 2026
    Review
  5. Article
  6. Spontaneous whole retinal degeneration in aged Beclin1 heterozygous mice.Journal of neural transmission (Vienna, Austria : 1996) · 2026
    Article
  7. Article
  8. Article
  9. Article
  10. Article
  11. Article
  12. Article
  13. Review
  14. Article
  15. Article
  16. Article
  17. Review
  18. Article
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

David S HansmanSchool of Biological Sciences, University of Adelaide, Adelaide, SA, Australia. Electronic address: david.hansman@adelaide.edu.au.
Jianhai DuDepartment of Ophthalmology and Visual Sciences, Department of Biochemistry and Molecular Medicine, West Virginia University, Morgantown, WV 26506, USA.
Robert J CassonDiscipline of Ophthalmology and Visual Science, Adelaide Medical School, University of Adelaide, Adelaide, SA, Australia.
Daniel J PeetSchool of Biological Sciences, University of Adelaide, Adelaide, SA, Australia.

Funding

Mitochondrial pyruvate transport in retinal health and diseaseR01EY031324 · NEI · WEST VIRGINIA UNIVERSITY · PI Jianhai Du · 2022 to 2024
$1.2M
Metabolic dysfunction from ECM remodeling in diseases of human RPER01EY034364 · UNIVERSITY OF WASHINGTON · 2025 to 2025
$543k
Proline metabolism in retinal healthR01EY032462 · WEST VIRGINIA UNIVERSITY · 2025 to 2025
$380k
NEI NIH HHS R01 EY031324NEI NIH HHS R01 EY031720NEI NIH HHS R01 EY032462NEI NIH HHS R01 EY034364NEI NIH HHS U01 EY034591
6 · The paper itself

Abstract

To meet the prodigious bioenergetic demands of the photoreceptors, glucose and other nutrients must traverse the retinal pigment epithelium (RPE), a polarised monolayer of cells that lie at the interface between the outer retina and the choroid, the principal vascular layer of the eye. Recent investigations have revealed a metabolic ecosystem in the outer retina where the photoreceptors and RPE engage in a complex exchange of sugars, amino acids, and other metabolites. Perturbation of this delicate metabolic balance has been identified in the aging retina, as well as in age-related macular degeneration (AMD), the leading cause of blindness in the Western world. Also common in the aging and diseased retina are elevated levels of cytokines, oxidative stress, advanced glycation end-products, increased growth factor signalling, and biomechanical stress - all of which have been associated with metabolic dysregulation in non-retinal cell types and tissues. Herein, we outline the role of these factors in retinal homeostasis, aging, and disease. We discuss their effects on glucose, mitochondrial, lipid, and amino acid metabolism in tissues and cell types outside the retina, highlighting the signalling pathways through which they induce these changes. Lastly, we discuss promising avenues for future research investigating the roles of these pathological conditions on retinal metabolism, potentially offering novel therapeutic approaches to combat age-related retinal disease.

Indexed as

AgingRetinal DiseasesRetinal Pigment EpitheliumAnimalsEnergy MetabolismHumansMacular DegenerationOxidative StressAge-related macular degenerationMetabolic ecosystemRetina inflammationRetinal pigment epitheliumRetina metabolismRPE agingRPE metabolism

Identifiers

PMID39433211
PMCPMC11833275

What Socratic holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.