Evidence map›Paper›PMID 39468595›Full record

ArticleJournal of translational medicine2024

Caerin 1.1 and 1.9 peptides halt B16 melanoma metastatic tumours via expanding cDC1 and reprogramming tumour macrophages.

Quanlan Fu, Yuandong Luo, Junjie Li, Hejie Li, Xiaosong Liu, Zhu Chen, Guoying Ni, Tianfang Wang

Abstract read
In one paragraph

Article in Journal of translational medicine, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Article
  2. Caerin 1.1/1.9 peptides controlFrontiers in microbiology · 2026
    Article
  3. Article
  4. Article
  5. Review
  6. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Quanlan FuMedical School of Guizhou University, Guiyang, Guizhou, 550000, China.
Yuandong LuoMedical School of Guizhou University, Guiyang, Guizhou, 550000, China.
Junjie LiThe First Affiliated Hospital/School of Clinical Medicine of Guangdong Pharmaceutical University, Guangzhou, 510080, China.
Hejie LiSchool of Science, Technology and Engineering, University of the Sunshine Coast, Maroochydore, BC, QLD 4558, Australia.
Xiaosong LiuMedical School of Guizhou University, Guiyang, Guizhou, 550000, China.
Zhu ChenGuiyang Hospital of Stomatology, Guiyang, Guizhou, 550004, China. zhuchen@gzu.edu.cn.
Guoying NiCancer Research Institute, First People's Hospital of Foshan, Foshan, Guangdong, 528000, China. ngy2003@gmail.com.
Tianfang WangSchool of Science, Technology and Engineering, University of the Sunshine Coast, Maroochydore, BC, QLD 4558, Australia. twang@usc.edu.au.ORCID 0000-0002-4876-7767

Funding

Dengfeng project of Foshan First People's Hospital 2019A008National Natural Science Foundation of China 31971355Natural Science Foundation of Guangdong Province 2020A1515010855
6 · The paper itself

Abstract

backgroundCancer immunotherapy, particularly immune checkpoint inhibitors (ICBs) such as anti-PD-1 antibodies, has revolutionised cancer treatment, although response rates vary among patients. Previous studies have demonstrated that caerin 1.1 and 1.9, host-defence peptides from the Australian tree frog, enhance the effectiveness of anti-PD-1 and therapeutic vaccines in a murine TC-1 model by activating tumour-associated macrophages intratumorally.

methodsWe employed a murine B16 melanoma model to investigate the therapeutic potential of caerin 1.1 and 1.9 in combination with anti-CD47 and a therapeutic vaccine (triple therapy, TT). Tumour growth of caerin-injected primary tumours and distant metastatic tumours was assessed, and survival analysis conducted. Single-cell RNA sequencing (scRNAseq) of CD45

resultsThe TT treatment significantly reduced tumour volumes on the treated side compared to untreated and control groups, with notable effects observed by Day 21. Survival analysis indicated extended survival in mice receiving TT, both on the treated and distant sides. scRNAseq revealed a notable expansion of conventional type 1 dendritic cells (cDC1s) and CD4

conclusionOur findings suggest that caerin 1.1 and 1.9, combined with immunotherapy, effectively modulate the tumour microenvironment in primary and secondary tumours, leading to reduced tumour growth and enhanced systemic immunity. Further investigation into these mechanisms could pave the way for improved combination therapies in advanced melanoma treatment.

Indexed as

Melanoma, ExperimentalMice, Inbred C57BLNeoplasm MetastasisTumor-Associated MacrophagesTumor MicroenvironmentAnimalsCD47 AntigenCell Line, TumorCellular ReprogrammingDendritic CellsMacrophagesMicePeptidesCD47 AntigenPeptidesanti-CD47 antibodyB16 cellCaerin peptideCD4+CD8+ T cellcDC1Cell-cell communicationImmunotherapyMacrophageMelanoma

Identifiers

PMID39468595
PMCPMC11514859

What Socratic holds

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LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.