Evidence mapPaperPMID 39468702Full record

ArticleBMC pharmacology & toxicology2024

Nephroprotective role of resveratrol in renal ischemia-reperfusion injury: a preclinical study in Sprague-Dawley rats.

Elaf R Alaasam, Ali M Janabi, Karrar M Al-Buthabhak, Rihab H Almudhafar, Najah R Hadi, Athanasios Alexiou, Marios Papadakis, Mohammed E Abo-El Fetoh, Dalia Fouad, Gaber El-Saber Batiha

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Article in BMC pharmacology & toxicology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Elaf R AlaasamAlsadar Medical City, Directorate of Najaf Health, Najaf, Iraq.
Ali M JanabiDepartment of Pharmacology and Toxicology, Faculty of Pharmacy, University of Kufa, Najaf, Iraq.
Karrar M Al-ButhabhakDepartment of Internal Medicine, Faculty of Medicine, University of Kufa, Najaf, Iraq.
Rihab H AlmudhafarDepartment of Pathology and Forensic Medicine, Faculty of Medicine, University of Kufa, Najaf, Iraq.
Najah R HadiDepartment of Pharmacology and Therapeutics, Faculty of Pharmacy, University of Kufa, Najaf, Iraq.
Athanasios AlexiouDepartment of Research & Development, Funogen, Athens, 11741, Greece, Attiki.
Marios PapadakisDepartment of Surgery II, University Hospital Witten-Herdecke, University of Witten-Herdecke, Heusnerstrasse 40, 42283, Wuppertal, Germany. drmariospapadakis@gmail.com.
Mohammed E Abo-El FetohDepartment of Pharmacology and Toxicology, Faculty of Pharmacy, Egyptian Russian University, Cairo, Egypt. mohamed-ezzat@eru.edu.eg.
Dalia FouadDepartment of Zoology, College of Science, King Saud University, PO Box 22452, Riyadh, 11495, Saudi Arabia.
Gaber El-Saber BatihaDepartment of Pharmacology and Therapeutics, Faculty of Veterinary Medicine, Damanhour University, Damanhour, AlBeheira, 22511, Egypt. dr_gaber_batiha@vetmed.dmu.edu.eg.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundRenal ischemia-reperfusion injury (IRI) is a significant contributor to renal dysfunction, acute kidney injury (AKI), and associated morbidity and mortality. Resveratrol, a polyphenol and phytoalexin, is known for its anti-inflammatory, antioxidant, and anti-cancer properties. This study investigates the nephroprotective potential of resveratrol in a rat model of renal IRI. MATERIALS AND

methodsTwenty-eight male Sprague-Dawley rats were divided into four groups: Sham, IRI, DMSO, and Resveratrol. The Sham group underwent identical procedures without renal pedicle clamping, while the IRI group experienced 30 min of ischemia followed by 2 h of reperfusion. The DMSO group received dimethyl sulfoxide (DMSO) intraperitoneally 30 min before ischemia, and the Resveratrol group received 30 mg/kg resveratrol intraperitoneally 30 min before ischemia. Biochemical parameters (Urea, creatinine, IL-1β, NF-κβ, SOD, GSH, Bcl-2, and caspase-3) and histopathological changes were assessed.

resultsIRI caused a substantial increase in serum creatinine, Urea, IL-1β, NF-κβ, and caspase-3 levels, while simultaneously decreasing SOD, GSH, and Bcl-2 levels. Resveratrol treatment mitigated these effects by lowering inflammatory and apoptotic markers, enhancing antioxidant defenses, and improving histological outcomes.

conclusionResveratrol demonstrates significant nephroprotective effects in renal IRI, primarily through its antioxidant, anti-inflammatory, and anti-apoptotic properties.

Indexed as

AntioxidantsKidneyRats, Sprague-DawleyReperfusion InjuryResveratrolAcute Kidney InjuryAnimalsCaspase 3CreatinineInterleukin-1betaMaleRatsAntioxidantsCaspase 3CreatinineInterleukin-1betaResveratrolApoptosisInflammationNephroprotectionOxidative StressRenal Ischemia-Reperfusion InjuryResveratrol

Identifiers

PMID39468702
PMCPMC11520524

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.