Evidence map›Paper›PMID 39516813›Full record

ArticleThe journal of headache and pain2024

Microbiota alterations are related to migraine food triggers and inflammatory markers in chronic migraine patients with medication overuse headache.

Doga Vuralli, Merve Ceren Akgor, Hale Gok Dagidir, Pınar Onat, Meltem Yalinay, Ugur Sezerman, Hayrunnisa Bolay

Abstract read
In one paragraph

Article in The journal of headache and pain, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed, 2 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 2 syntheses or guidelines pooled it.

  1. Pooled it
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  12. The effect of short-term consumption ofExperimental biology and medicine (Maywood, N.J.) · 2026
    Article
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  14. Article
  15. Article
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  17. The Gut Microbiome and Migraine: Updates in Understanding.Current neurology and neuroscience reports · 2025
    Review
  18. Review
  19. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Doga VuralliDepartment of Neurology and Algology, Gazi University Faculty of Medicine, Ankara, Türkiye.
Merve Ceren AkgorNeuroscience and Neurotechnology Center of Excellence (NÖROM), Gazi University, Ankara, Türkiye.
Hale Gok DagidirNeuroscience and Neurotechnology Center of Excellence (NÖROM), Gazi University, Ankara, Türkiye.
Pınar OnatEpigenetiks Genetic Bioinformatics Software Inc., Istanbul, Türkiye.
Meltem YalinayDepartment of Clinical Microbiology, Gazi University Faculty of Medicine, Ankara, Türkiye.
Ugur SezermanDepartment of Biostatistics and Medical Informatics, Acibadem University Faculty of Medicine, Istanbul, Türkiye.
Hayrunnisa BolayDepartment of Neurology and Algology, Gazi University Faculty of Medicine, Ankara, Türkiye. hbolay@gazi.edu.tr.

Funding

Gazi University ADEP project grant TGA-2022-7857
6 · The paper itself

Abstract

objectiveChronic migraine (CM) patients with medication overuse headache (MOH) were recently shown to be associated with leaky gut and inflammation. We aimed to investigate gut microbiota profiles of CM patients with MOH, and their correlations with inflammatory serum parameters, migraine food triggers, and comorbid anxiety and depression. MATERIALS AND

methodsThe study included women participants (32 CM patients with NSAID overuse headache, and 16 healthy non-headache sufferers). Migraine duration, monthly migraine headache days, presence of irritable bowel syndrome symptoms, and HADS-D and HADS-A scores were recorded. Serum samples were collected to measure circulating LPS, HMGB1, HIF-1α, and IL-6. The gut microbiota profiles of the patients were evaluated using fecal samples.

resultsSerum LPS, HMGB1, HIF-1α, and IL-6 levels were significantly higher in the CM + MOH group compared to the healthy controls. HADS-A and HADS-D scores were considerably higher in the CM + MOH group compared to the healthy controls. In the microbiota analysis, alpha and beta diversities were similar between the two groups. The class Clostridia, the order Eubacteriales, and the genus Ruminococcus were less abundant in the CM + NSAID overuse headache group compared to the control group. At the genus level Desulfovibrio, Gemmiger, and Dialister and at the species level, Clostridium fessum, Blautia luti, Dorea longicatena, Eubacterium coprostanoligenes, and Gemmiger formicilis were more abundant in the CM + NSAID overuse headache group compared to the control group. Desulfovibrio, Gemmiger, Dialister, Ethanoligenens harbinense, Eubacterium coprostanoligenes, Dorea longicatena, and Thermoclostridium stercorarium showed positive correlations and Clostridia bacteria showed negative correlations with migraine food triggers. Positive correlations were found between LPS and Hapalosiphonaceae, HMGB1 and Melghirimyces, HIF1-α and Rouxeilla and Blautia luti, IL-6 and Melghirimyces and Ruminococcus.

conclusionIn CM patients with MOH, we have revealed the presence of dysbiosis towards an inflammatory state, and positive correlations were shown between altered gut microbiota and inflammatory serum parameters and migraine food triggers.

Indexed as

Gastrointestinal MicrobiomeHeadache Disorders, SecondaryMigraine DisordersAdultAnti-Inflammatory Agents, Non-SteroidalBiomarkersFemaleFoodHumansInflammationMiddle AgedAnti-Inflammatory Agents, Non-SteroidalBiomarkersChronic migraineHIF-1αHMGB1InflammationLeaky gutLipopolysaccharideMedication overuse headacheMicrobiota

Identifiers

PMID39516813
PMCPMC11546420

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.