Evidence mapPaperPMID 39627610Full record

ArticleCellular & molecular immunology2025

Dysregulation in keratinocytes drives systemic lupus erythematosus onset.

Jingru Tian, Liqing Shi, Dingyao Zhang, Xu Yao, Ming Zhao, Snehlata Kumari, Jun Lu, Di Yu, Qianjin Lu

Abstract read
In one paragraph

Article in Cellular & molecular immunology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 1 synthesis or guideline pooled it.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Jingru Tian *Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences and Peking Union Medical College, Nanjing, China.
Liqing Shi *Hospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences and Peking Union Medical College, Nanjing, China.
Dingyao ZhangProgram of Computational Biology and Bioinformatics, Yale University, New Haven, CT, USA.
Xu YaoHospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences and Peking Union Medical College, Nanjing, China.
Ming ZhaoHospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences and Peking Union Medical College, Nanjing, China.
Snehlata KumariFaculty of Medicine, Frazer Institute, The University of Queensland, Brisbane, QLD, Australia.ORCID http://orcid.org/0000-0002-4971-3162
Jun LuYale Stem Cell Center, New Haven, CT, USA.
Di YuFaculty of Medicine, Frazer Institute, The University of Queensland, Brisbane, QLD, Australia.ORCID http://orcid.org/0000-0003-1721-8922
Qianjin LuHospital for Skin Diseases, Institute of Dermatology, Chinese Academy of Medical Sciences and Peking Union Medical College, Nanjing, China. qianlu5860@pumcderm.cams.cn.ORCID http://orcid.org/0000-0002-7749-9618

Funding

National Natural Science Foundation of China (National Science Foundation of China) 81830097National Natural Science Foundation of China (National Science Foundation of China) 82203933
6 · The paper itself

Abstract

Systemic lupus erythematosus (SLE) is a complex, multiorgan autoimmune disorder. Although it is widely believed that SLE originates from immune cell dysregulation, the etiology of SLE is not yet clear. Here, we propose a new theory in which SLE can be directly initiated by molecular alterations in keratinocytes rather than immune cells. We found that the level of peroxisome proliferator-activated receptor gamma (PPARγ) is substantially reduced in the skin lesions of patients, and replicating this reduction in mice led to rapid disease onset with multiple hallmarks of SLE. As PPARγ decreases in keratinocytes, which is accompanied by increased occupancy of interferon regulatory factor 3 at the type I interferon locus, dendritic cells (DCs) are recruited to the epidermis and are activated by keratinocyte-secreted type I interferon. These activated DCs migrate to local draining lymph nodes, where they activate CD4

Indexed as

Dendritic CellsKeratinocytesLupus Erythematosus, SystemicPPAR gammaAnimalsCD4-Positive T-LymphocytesCell DifferentiationDisease Models, AnimalFemaleHumansInterferon Type IMiceMice, Inbred C57BLInterferon Type IPPAR gammaDendritic cellsInterferon regulatory factor 3KeratinocytesPeroxisome proliferator-activated receptor gammaSystemic lupus erythematosusType I interferon

Identifiers

PMID39627610
PMCPMC11686216

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.