Evidence map›Paper›PMID 39628485›Full record

ArticleFrontiers in immunology2024

Stimulation of microneedles alleviates pathology of Parkinson's disease in mice by regulating the CD4+/CD8+ cells from the periphery to the brain.

Jin Hee Kim, Yujin Choi, Jin Se Kim, Hanbyeol Lee, In Gyoung Ju, Na Young Yoo, Sookie La, Do Hyeon Jeong, Changsu Na, Hi-Joon Park and 1 more

Abstract read
In one paragraph

Article in Frontiers in immunology, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Jin Hee KimDepartment of Biomedical and Pharmaceutical Sciences, Kyung Hee University, Seoul, Republic of Korea.
Yujin ChoiDepartment of Biomedical and Pharmaceutical Sciences, Kyung Hee University, Seoul, Republic of Korea.
Jin Se KimDepartment of Biomedical and Pharmaceutical Sciences, Kyung Hee University, Seoul, Republic of Korea.
Hanbyeol LeeDepartment of Biomedical and Pharmaceutical Sciences, Kyung Hee University, Seoul, Republic of Korea.
In Gyoung JuDepartment of Oriental Pharmaceutical Science and Kyung Hee East-West Pharmaceutical Research Institute, College of Pharmacy, Kyung Hee University, Seoul, Republic of Korea.
Na Young YooRaphas Co. Ltd., Seoul, Republic of Korea.
Sookie LaRaphas Co. Ltd., Seoul, Republic of Korea.
Do Hyeon JeongRaphas Co. Ltd., Seoul, Republic of Korea.
Changsu NaDepartment of Acupoint and Meridian, Korean Medical College, Dongshin University, Naju, Republic of Korea.
Hi-Joon ParkDepartment of Science in Korean Medicine, College of Korean Medicine, Kyung Hee University, Seoul, Republic of Korea.
Myung Sook OhDepartment of Biomedical and Pharmaceutical Sciences, Kyung Hee University, Seoul, Republic of Korea.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Introduction: Immune dysfunction is a major cause of neuroinflammation and accelerates the progression of Parkinson's disease (PD). Numerous studies have shown that stimulation of specific acupuncture points (acupoints) can ameliorate PD symptoms. The purpose of this study was to investigate whether attaching microneedles to acupoints would improve PD pathology by recovering immune dysfunction. Methods: The PD mouse model was induced by intrastriatal injection of 6-hydroxydopamine (6-OHDA), and microneedle patches (MPs) or sham patches (SPs) were attached to GB20 and GB34, representative acupoints for treating PD for 14 days. Results: First, the behavioral experiment showed that motor disorders induced by 6-OHDA were significantly improved by MP. Simultaneously, 6-OHDA-induced dopaminergic neuronal death and brain neuroinflammation decreased. Conversely, SP had no effect on behavioral disorders, neuronal death, or neuroinflammation. Measurement results from flow cytometry of immune cells in the brain and blood revealed a disruption in the CD4+/CD8+ ratio in the 6-OHDA group, which was significantly restored in the MP group. The brain mRNA expression of cytokines was significantly increased in the 6-OHDA group, which was significantly decreased by MP. Discussion: Overall, our results suggest that the attachment of MPs to GB20 and GB34 is a new method to effectively improve the pathology of PD by restoring peripheral and brain immune function.

Indexed as

BrainCD8-Positive T-LymphocytesDisease Models, AnimalParkinson DiseaseAcupuncture PointsAcupuncture TherapyAnimalsCD4-Positive T-LymphocytesCytokinesMaleMiceMice, Inbred C57BLNeedlesOxidopamineCytokinesOxidopamineacupuncture pointmicroneedleneuroinflammationParkinson’s diseaseperipheral immune

Identifiers

PMID39628485
PMCPMC11611716

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.