Evidence map›Paper›PMID 39672896›Full record

ReviewNature aging2024

Hallmarks of female reproductive aging in physiologic aging mice.

Julia L Balough, Shweta S Dipali, Karen Velez, T Rajendra Kumar, Francesca E Duncan

Abstract readReview
In one paragraph

Review in Nature aging, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed.

  1. Article
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  7. Review
  8. Article
  9. Article
  10. Review
  11. Article
  12. Article
  13. Review
  14. Female Aging Affects Coilin Pattern in Mouse Cumulus Cells.Journal of developmental biology · 2026
    Article
  15. Review
  16. Review
  17. Review
  18. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Julia L Balough *Center for Reproductive Longevity and Equality, Buck Institute for Research on Aging, Novato, CA, USA.ORCID 0000-0002-5711-440X
Shweta S Dipali *Department of Obstetrics and Gynecology, Feinberg School of Medicine, Northwestern University, Chicago, IL, USA.ORCID 0000-0002-4964-4822
Karen Velez *Department of Obstetrics and Gynecology, Feinberg School of Medicine, Northwestern University, Chicago, IL, USA.
T Rajendra KumarDepartment of Obstetrics and Gynecology, University of Colorado Anschutz Medical Campus, Aurora, CO, USA.
Francesca E DuncanCenter for Reproductive Longevity and Equality, Buck Institute for Research on Aging, Novato, CA, USA. f-duncan@northwestern.edu.ORCID 0000-0002-3756-9394

Funding

The Aging Pituitary/Gonadal AxisP01AG029531 · NIA · WICHITA STATE UNIVERSITY · PI GEORGE R BOUSFIELD · 2009 to 2026
$25.9M
FSH Glycoforms and Ovarian Signaling PathwaysR01HD103384 · NICHD · UNIVERSITY OF COLORADO DENVER · PI KUMAR, T. RAJENDRA · 2021 to 2025
$2.8M
Oocyte genomic instability as a driver of the aging ovarian innate immune responseR01HD105752 · NICHD · NORTHWESTERN UNIVERSITY AT CHICAGO · PI DUNCAN, FRANCESCA E., GERTON, JENNIFER L · 2021 to 2025
$2.4M
Northwestern Center for Reproductive Science Predoctoral Training Program in Reproductive Science, Medicine, and TechnologyT32HD094699 · NICHD · NORTHWESTERN UNIVERSITY AT CHICAGO · PI Ji-Yong Julie Kim · 2019 to 2026
$1.3M
Gonadal and extra-gonadal actions of FSH glycoforms in agingR56AG056046 · NIA · UNIVERSITY OF COLORADO DENVER · PI KUMAR, T. RAJENDRA · 2017 to 2017
$481k
NIA NIH HHS P01 AG029531NIA NIH HHS R56 AG056046NICHD NIH HHS R01 HD103384NICHD NIH HHS R01 HD105752NICHD NIH HHS T32 HD094699U.S. Department of Health & Human Services | NIH | Eunice Kennedy Shriver National Institute of Child Health and Human Development (NICHD) R01HD103384U.S. Department of Health & Human Services | NIH | Eunice Kennedy Shriver National Institute of Child Health and Human Development (NICHD) R01HD105752U.S. Department of Health & Human Services | NIH | National Institute on Aging (U.S. National Institute on Aging) AG029531U.S. Department of Health & Human Services | NIH | National Institute on Aging (U.S. National Institute on Aging) AG056046
6 · The paper itself

Abstract

The female reproductive axis is one of the first organ systems to age, which has consequences for fertility and overall health. Here, we provide a comprehensive overview of the biological process of female reproductive aging across reproductive organs, tissues and cells based on research with widely used physiologic aging mouse models, and describe the mechanisms that underpin these phenotypes. Overall, aging is associated with dysregulation of the hypothalamic-pituitary-ovarian axis, perturbations of the ovarian stroma, reduced egg quantity and quality, and altered uterine morphology and function that contributes to reduced capacity for fertilization and impaired embryo development. Ultimately, these age-related phenotypes contribute to altered pregnancy outcomes and adverse consequences in offspring. Conserved mechanisms of aging, as well as those unique to the reproductive system, underlie these phenotypes. The knowledge of such mechanisms will lead to development of therapeutics to extend female reproductive longevity and support endocrine function and overall health.

Indexed as

AgingReproductionAnimalsFemaleHypothalamo-Hypophyseal SystemMiceOvaryPregnancyUterus

Identifiers

PMID39672896
PMCPMC12139273

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.