Evidence mapPaperPMID 39676616Full record

ReviewJournal of diabetes2024

Autophagy modulators in type 2 diabetes: A new perspective.

Ayah Talal Zaidalkilani, Hayder M Al-Kuraishy, Esraa H Fahad, Ali I Al-Gareeb, Yaser Hosny Ali Elewa, Mahmoud Hosny Zahran, Athanasios Alexiou, Marios Papadakis, Ammar Al-Farga, Gaber El-Saber Batiha

Abstract readReview
In one paragraph

Review in Journal of diabetes, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

  1. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Ayah Talal ZaidalkilaniDepartment of Nutrition, Faculty of Pharmacy and Medical Sciences, University of Petra, Amman, Jordan.
Hayder M Al-KuraishyDepartment of Clinical Pharmacology and Medicine, College of Medicine, Al-Mustansiriyah University, Baghdad, Iraq.
Esraa H FahadDepartment of Pharmacology and Toxicology, College of Pharmacy, Mustansiriyah University, Baghdad, Iraq.
Ali I Al-GareebDepartment of Clinical Pharmacology and Medicine, College of Medicine, Al-Mustansiriyah University, Baghdad, Iraq.
Yaser Hosny Ali ElewaDepartment of Histology and Cytology, Faculty of Veterinary Medicine, Zagazig University, Zagazig, Egypt.ORCID https://orcid.org/0000-0002-7347-4587
Mahmoud Hosny ZahranInternal Medicine Department, Faculty of Medicine, Zagazig University, Zagazig, Egypt.ORCID https://orcid.org/0000-0003-2108-0604
Athanasios AlexiouUniversity Centre for Research & Development, Chandigarh University, Mohali, Punjab, India.ORCID https://orcid.org/0000-0002-2206-7236
Marios PapadakisDepartment of Surgery II, University Hospital Witten-Herdecke, Wuppertal, Germany.
Ammar Al-FargaDepartment of Biochemistry, College of Science University of Jeddah, Jeddah, Saudi Arabia.
Gaber El-Saber BatihaDepartment of Pharmacology and Therapeutics, Faculty of Veterinary Medicine, Damanhur University, Damanhur, AlBeheira, Egypt.ORCID https://orcid.org/0000-0002-7817-425X

Funding

Projekt DEALUniversity of Witten-Herdecke
6 · The paper itself

Abstract

Type 2 diabetes (T2D) is a chronic metabolic disorder caused by defective insulin signaling, insulin resistance, and impairment of insulin secretion. Autophagy is a conserved lysosomal-dependent catabolic cellular pathway involved in the pathogenesis of T2D and its complications. Basal autophagy regulates pancreatic β-cell function by enhancing insulin release and peripheral insulin sensitivity. Therefore, defective autophagy is associated with impairment of pancreatic β-cell function and the development of insulin rersistance (IR). However, over-activated autophagy increases apoptosis of pancreatic β-cells leading to pancreatic β-cell dysfunction. Hence, autophagy plays a double-edged sword role in T2D. Therefore, the use of autophagy modulators including inhibitors and activators may affect the pathogenesis of T2D. Hence, this review aims to clarify the potential role of autophagy inhibitors and activators in T2D.

Indexed as

AutophagyDiabetes Mellitus, Type 2Insulin-Secreting CellsAnimalsApoptosisHumansInsulinInsulin ResistanceSignal TransductionInsulinautophagyinsulin resistancetype 2 diabetes mellitusβ‐cells

Identifiers

PMID39676616
PMCPMC11647182

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.