Evidence map›Paper›PMID 39683799›Full record

ArticleMolecules (Basel, Switzerland)2024

M2BPgs-HCC: An Automated Multilectin Bead Array Indicating Aberrant Glycosylation Signatures Toward Hepatitis C Virus-Associated Hepatocellular Carcinoma Prognosis.

Hiroko Shimazaki, Haruki Uojima, Kazumi Yamasaki, Tomomi Obayashi, Sayaka Fuseya, Takashi Sato, Masashi Mizokami, Atsushi Kuno

Abstract read
In one paragraph

Article in Molecules (Basel, Switzerland), 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Hiroko ShimazakiMolecular & Cellular Glycoproteomics Research Group, Cellular and Molecular Biotechnology Research Institute, National Institute of Advanced Industrial Science & Technology, Tsukuba 305-8565, Japan.
Haruki UojimaGenome Medical Science Project, National Center for Global Health and Medicine, Ichikawa 272-8516, Japan.ORCID 0000-0003-1719-1352
Kazumi YamasakiClinical Research Center, NHO National Hospital Organization Nagasaki Medical Center, Omura 856-0835, Japan.ORCID 0000-0001-8876-4440
Tomomi ObayashiMolecular & Cellular Glycoproteomics Research Group, Cellular and Molecular Biotechnology Research Institute, National Institute of Advanced Industrial Science & Technology, Tsukuba 305-8565, Japan.
Sayaka FuseyaMolecular & Cellular Glycoproteomics Research Group, Cellular and Molecular Biotechnology Research Institute, National Institute of Advanced Industrial Science & Technology, Tsukuba 305-8565, Japan.ORCID 0000-0002-0963-9949
Takashi SatoMolecular & Cellular Glycoproteomics Research Group, Cellular and Molecular Biotechnology Research Institute, National Institute of Advanced Industrial Science & Technology, Tsukuba 305-8565, Japan.ORCID 0000-0002-9973-8923
Masashi MizokamiGenome Medical Science Project, National Center for Global Health and Medicine, Ichikawa 272-8516, Japan.ORCID 0000-0001-5269-4006
Atsushi KunoMolecular & Cellular Glycoproteomics Research Group, Cellular and Molecular Biotechnology Research Institute, National Institute of Advanced Industrial Science & Technology, Tsukuba 305-8565, Japan.ORCID 0000-0002-6147-6171

Funding

Japan Science and Technology Agency JPMJTR204AJapan Society for the Promotion of Science 23H02680
6 · The paper itself

Abstract

Regular monitoring of patients with a history of hepatitis C virus (HCV) infection is critical for the detection and management of hepatocellular carcinoma (HCC). Mac-2 binding protein glycosylation isomer (M2BPGi) has been used to monitor fibrosis progression and predict HCC. However, HCC prediction based on M2BPGi has not been optimized. Here, we identified HCC risk-related glycan signatures of M2BP using a newly developed automated bead array with multiplexed lectins. Among 955 patients with HCV who achieved sustained virological response following direct-acting antiviral treatment, we compared M2BP glycosylation from sera of 42 patients diagnosed with HCC during follow-up and 43 without HCC (control) by the lectin microarray. At the HCC observation point, we found significant differences in 17 lectins. Using an automated bead array with 12 of 17 lectins, a principal component analysis (PCA) biplot differentiated HCC from control, along the PC1 axis, explaining 75.2% of variance. Based on PC1, we generated a scoring formula for an HCC-related glycosylation signature on M2BP (M2BPgs-HCC), showing good diagnostic performance for HCC (

Indexed as

Carcinoma, HepatocellularHepacivirusLectinsLiver NeoplasmsAgedAntigens, NeoplasmBiomarkers, TumorFemaleGlycosylationHepatitis CHumansMaleMembrane GlycoproteinsMiddle AgedPrognosisAntigens, NeoplasmBiomarkers, TumorLectinsMembrane GlycoproteinsTAA90K protein, humanglycosylationhepatocellular carcinomalectin arrayM2BPGi

Identifiers

PMID39683799
PMCPMC11643838

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.