ArticleAmerican journal of physiology. Renal physiology2025
Intestinal barrier function declines during polycystic kidney disease progression.
Randee Sedaka, Caleb Lovelady, Emily Hallit, Branden Duyvestyn, Sejal Shinde, Aida Moran-Reyna, Goo Lee, Shinobu Yamaguchi, Craig L Maynard, Takamitsu Saigusa
Abstract read
In one paragraphArticle in American journal of physiology. Renal physiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
0numbers the graph read from it
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0citing papers in PubMed
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1 · What the graph read from itWhat it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registryThe trial behind it
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3 · Its place in the literatureWho cites it
0 citing papers in PubMed.
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4 · The recordCorrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what moneyAuthors and funding
10 authors.
Randee SedakaSection of Cardio-Renal Physiology and Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Alabama, United States.
Caleb LoveladySection of Cardio-Renal Physiology and Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Alabama, United States.
Emily HallitSection of Cardio-Renal Physiology and Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Alabama, United States.
Branden DuyvestynSection of Cardio-Renal Physiology and Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Alabama, United States.
Sejal ShindeSection of Cardio-Renal Physiology and Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Alabama, United States.
Aida Moran-ReynaSection of Cardio-Renal Physiology and Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Alabama, United States.
Goo LeeDivision of Anatomic Pathology, Department of Pathology, University of Alabama at Birmingham, Alabama, United States.ORCID 0000-0003-3025-5234 Shinobu YamaguchiSection of Cardio-Renal Physiology and Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Alabama, United States.
Craig L MaynardDivision of Molecular & Cellular Pathology, Department of Pathology, University of Alabama at Birmingham, Alabama, United States.ORCID 0000-0001-5876-8168 Takamitsu SaigusaSection of Cardio-Renal Physiology and Medicine, Division of Nephrology, Department of Medicine, University of Alabama at Birmingham, Alabama, United States.ORCID 0000-0002-2398-1572 Funding
TRAINING IN CELLULAR PHYSIOLOGY MEMBRANE TRANSPORTT32DK007545 · UNIVERSITY OF ALABAMA AT BIRMINGHAM · 1987 to 2005
$660kThe Role of Wheat-Gluten in Polycystic Kidney DiseaseR01DK132028 · UNIVERSITY OF ALABAMA AT BIRMINGHAM · 2025 to 2025
$297kHHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) DK132028HHS | NIH | NIDDK | Division of Diabetes, Endocrinology, and Metabolic Diseases (DEM) DK007545NIDDK NIH HHS R01 DK132028NIDDK NIH HHS T32 DK007545
6 · The paper itselfAbstract
Most patients with autosomal dominant polycystic kidney disease (ADPKD) develop kidney cysts due to germline
Indexed as
Intestinal MucosaKidneyPolycystic Kidney, Autosomal DominantTRPP Cation ChannelsAnimalsDisease Models, AnimalDisease ProgressionIntestinal Barrier FunctionMaleMiceMice, KnockoutPermeabilityTRPP Cation ChannelsADPKDcolonintegritymotilitywater
Identifiers
PMID39694536
PMCPMC12176450
What Socratic holds
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