Evidence mapPaperPMID 39707389Full record

ArticleOrphanet journal of rare diseases2024

Clinical differential factors in patients with hereditary transthyretin amyloidosis with Val142Ile and Ser43Asn mutations.

Sandra Milena Castellar-Leones, Edicson Ruiz-Ospina, Jorge Diaz-Ruiz, Cristian Correa-Arrieta, Xiomara Ruiz-Cortés, Diana Luzuriaga-Carpio, Dario Zambrano-Vera, Jeanneth Cedeño-Quincha, Luis Guerrero-Cepeda, Daniel César-Chávez and 1 more

Abstract readMulticenter Study
In one paragraph

Article in Orphanet journal of rare diseases, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

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0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Sandra Milena Castellar-LeonesDepartment of Physical Medicine and Rehabilitation, Facultad de Medicina, Universidad Nacional de Colombia, Carrera 30 No.45-03. Edificio 471, Piso 5to, Of. 513-A, Bogotá, Colombia. smcastellarl@unal.edu.co.ORCID 0000-0002-4559-2965
Edicson Ruiz-OspinaDepartment of Physical Medicine and Rehabilitation, Facultad de Medicina, Universidad Nacional de Colombia, Carrera 30 No.45-03. Edificio 471, Piso 5to, Of. 513-A, Bogotá, Colombia.
Jorge Diaz-RuizDepartment of Physical Medicine and Rehabilitation, Facultad de Medicina, Universidad Nacional de Colombia, Carrera 30 No.45-03. Edificio 471, Piso 5to, Of. 513-A, Bogotá, Colombia.
Cristian Correa-ArrietaResearch Center for Physiatry and Electrodiagnostics, (CIFEL), Bogotá, Colombia.
Xiomara Ruiz-CortésCenter of Rehabilitation RecuperAMI, Caquetá, Colombia.
Diana Luzuriaga-CarpioHospital General Manuel Ygnacio Monteros-IESS, Loja, Ecuador.
Dario Zambrano-VeraHospital de Especialidades Carlos Andrade Marín-IESS, Quito, Ecuador.
Jeanneth Cedeño-QuinchaHospital de Especialidades Eugenio Espejo, Quito, Ecuador.
Luis Guerrero-CepedaHospital de Especialidades Eugenio Espejo, Quito, Ecuador.
Daniel César-ChávezHospital Teodoro Maldonado Carbo-IESS, Guayaquil, Ecuador.
Fernando Ortiz-CorredorDepartment of Physical Medicine and Rehabilitation, Facultad de Medicina, Universidad Nacional de Colombia, Carrera 30 No.45-03. Edificio 471, Piso 5to, Of. 513-A, Bogotá, Colombia.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundHereditary transthyretin amyloidosis (hATTR) is a rare autosomal dominant disease with high clinical variability, influenced by both genotype and the geographic origins of carriers. There is a limited understanding of the Val142Ile and Ser43Asn recognised mutations in Ecuador and Colombia. Therefore, the objective of this study is to describe the neurological and functional characteristics of patients with hATTR associated with the Val142Ile and Ser43Asn mutations, as well as to identify possible differentiating factors between the two mutations.

methodsThis cross-sectional, multicenter study included 35 hATTR patients from rehabilitation centers in Ecuador and Colombia. Patients had confirmed Val142Ile or Ser43Asn mutations. Neurological and functional assessments included the Neurological Impairment Scale, Norfolk Quality of Life-Diabetic Neuropathy (QOL-DN), Composite Autonomic Symptom Score-31, and various motor function tests as nine-hole peg test (NHP). Quantitative Sensory Testing (QST) evaluating small fiber function, while ultrasound measured the cross-sectional area (CSA) of peripheral nerves. Statistical analysis employed nonparametric tests and random forest classifiers, using SHAP values to identify differentiating variables.

resultsVal142Ile carriers showed lower performance in the right NHP test and greater sensitivity to cold pain in hand and leg. Ultrasound revealed increased CSA of the median nerve at the elbow and arm and the ulnar nerve at the arm in Val142Ile carriers compared to Ser43Asn carriers. The final random forest model identified the NHP test, Norfolk QOL-DN score, and CSA of the median and ulnar nerves as key discriminating variables.

conclusionThis study identified significant neurophysiological and ultrasound markers differentiating Val142Ile and Ser43Asn mutations in hATTR-PN patients. Increased nerve CSA and specific motor and sensory impairments highlight the need for comprehensive evaluations to guide diagnosis and treatment.

Indexed as

Amyloid Neuropathies, FamilialMutationPrealbuminAdultAgedCross-Sectional StudiesFemaleHumansMaleMiddle AgedQuality of LifePrealbuminComposite Autonomic Symptom Score 31Hereditary transthyretin amyloidosisNerve cross-sectional areaNerve ultrasoundNeuropathy Impairment ScoreQuantitative sensory testing

Identifiers

PMID39707389
PMCPMC11662437

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.