Evidence map›Paper›PMID 39713070›Full record

ArticleWorld journal of experimental medicine2024

Statins decrease the risk of hepatocellular carcinoma in metabolic dysfunction-associated steatotic liver disease: A systematic review and meta-analysis.

Zahid Ijaz Tarar, Umer Farooq, Faisal Inayat, Sanket D Basida, Faisal Ibrahim, Mustafa Gandhi, Gul Nawaz, Arslan Afzal, Ammad J Chaudhary, Faisal Kamal and 2 more

Abstract read
In one paragraph

Article in World journal of experimental medicine, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
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  3. Review
  4. Review
  5. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Zahid Ijaz TararDepartment of Gastroenterology and Hepatology, University of Missouri, Columbia, MO 65212, United States.
Umer FarooqDepartment of Gastroenterology and Hepatology, St. Louis University, St. Louis, MO 63104, United States.
Faisal InayatDepartment of Internal Medicine, Allama Iqbal Medical College, Lahore, Punjab 54550, Pakistan. faisalinayat@hotmail.com.
Sanket D BasidaDepartment of Internal Medicine, University of Missouri, Columbia, MO 65212, United States.
Faisal IbrahimDepartment of Internal Medicine, Wexham Park Hospital, Wexham SL24HL, Slough, United Kingdom.
Mustafa GandhiDepartment of Gastroenterology and Hepatology, University of Missouri, Columbia, MO 65212, United States.
Gul NawazDepartment of Internal Medicine, Allama Iqbal Medical College, Lahore, Punjab 54550, Pakistan.
Arslan AfzalDepartment of Hospital Medicine, ECU Health Medical Center, Greenville, NC 27834, United States.
Ammad J ChaudharyDepartment of Internal Medicine, Henry Ford Hospital, Detroit, MI 48202, United States.
Faisal KamalDepartment of Gastroenterology and Hepatology, Thomas Jefferson University, Philadelphia, PA 19107, United States.
Ahmad H AliDepartment of Gastroenterology and Hepatology, University of Missouri, Columbia, MO 65212, United States.
Yezaz A GhouriDepartment of Gastroenterology and Hepatology, University of Missouri, Columbia, MO 65212, United States.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundMetabolic dysfunction-associated steatotic liver disease (MASLD) is a leading cause of chronic liver disease with a significant risk of developing hepatocellular carcinoma (HCC). Recent clinical evidence indicates the potential benefits of statins in cancer chemoprevention and therapeutics. However, it is still unclear if these drugs can lower the specific risk of HCC among patients with MASLD.

aimTo investigate the impact of statin use on the risk of HCC development in patients with MASLD.

methodsA systematic review and meta-analysis of all the studies was performed that measured the effect of statin use on HCC occurrence in patients with MASLD. The difference in HCC risk between statin users and non-users was calculated among MASLD patients. We also evaluated the risk difference between lipophilic versus hydrophilic statins and the effect of cumulative dose on HCC risk reduction.

resultsA total of four studies consisting of 291684 patients were included. MASLD patients on statin therapy had a 60% lower pooled risk of developing HCC compared to the non-statin group [relative risk (RR) = 0.40, 95%CI: 0.31-0.53,

conclusionStatin use lowers the risk of HCC in patients with MASLD. The higher cDDD and lipophilicity of statins correlate with the HCC risk reduction.

Indexed as

Hepatocellular carcinomaHydrophilic statinLipophilic statinMeta-analysisMetabolic dysfunction-associated steatotic liver diseaseStatins

Identifiers

PMID39713070
PMCPMC11551700

What Socratic holds

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LicenceCC BY-NC
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.