ArticleNature communications2024
Neutral drift upon threshold-like selection promotes variation in antibiotic resistance phenotype.
Article in Nature communications, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
4 citing papers in PubMed.
- Protein-protein interaction network architecture of human polygenic traits reveals domain-spanning connectivity and evolutionary pressures.Genome biology · 2026Article
- Molecular characterization of extended-spectrum beta-lactamase-producing Escherichia coli among children and farm animals in Agogo, Ghana.BMC microbiology · 2026Article
- The Constrained Disorder Principle: A Paradigm Shift for Accurate Interactome Mapping and Information Analysis in Complex Biological Systems.Bioengineering (Basel, Switzerland) · 2025Review
- Measuring differential fitness costs and interactions between genetic cassettes using fluorescent spectrophotometry.Applied and environmental microbiology · 2024Article
Corrections and comments
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Authors and funding
10 authors.
Funding
Abstract
Heritable phenotypic variation plays a central role in evolution by conferring rapid adaptive capacity to populations. Mechanisms that can explain genetic diversity by describing connections between genotype and organismal fitness have been described. However, the difficulty of acquiring comprehensive data on genotype-phenotype-environment relationships has hindered the efforts to explain how the ubiquitously observed phenotypic variation in populations emerges and is maintained. To address this challenge, we establish an experimental system where we can examine the genotype-phenotype relationships in a controlled environment. We perform long-term experimental evolution on VIM-2 β-lactamase, an antibiotic-resistance enzyme, to explore the conditions that promote the emergence and maintenance of phenotypic variation. We found that evolution in a static environment with low antibiotic concentrations can promote and maintain significant phenotypic variation within populations. Notably, evolution of VIM-2 under selection with a low antibiotic concentration led to variants that conferred resistance to over 100-fold higher antibiotic concentrations than used in selection. A model based on the previously described threshold-like relationship between enzyme phenotype and fitness generated using VIM-2's all single amino acid variants, sufficiently explains the emergence of standing phenotypic variation under static environmental conditions. Overall, our approach provides a tractable model for studying phenotypic variation and evolvability at the population level.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.