Evidence mapPaperPMID 39739648Full record

ArticleAdvanced science (Weinheim, Baden-Wurttemberg, Germany)2025

The Gut Microbiota Affects Anti-TNF Responsiveness by Activating the NAD

Jing Lei, Lin Lv, Li Zhong, Feng Xu, Wenhao Su, Yan Chen, Zhixuan Wu, Song He, Yongyu Chen

Abstract read
In one paragraph

Article in Advanced science (Weinheim, Baden-Wurttemberg, Germany), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed.

  1. Article
  2. Article
  3. NADAdvanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026
    Article
  4. Review
  5. Article
  6. Review
  7. Review
  8. Review
  9. MedComm · 2025
    Review
  10. Review
  11. The Gut Microbiota Affects Anti-TNF Responsiveness by Activating the NADAdvanced science (Weinheim, Baden-Wurttemberg, Germany) · 2025
    Article
  12. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Jing LeiDepartment of Gastroenterology, The Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400010, China.ORCID https://orcid.org/0000-0002-4577-4662
Lin LvDepartment of Gastroenterology, The Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400010, China.
Li ZhongDepartment of Gastroenterology, The Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400010, China.
Feng XuDepartment of Gastroenterology, The Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400010, China.
Wenhao SuDepartment of Gastroenterology, Renmin Hospital of Wuhan University, Hubei, 430060, China.
Yan ChenDepartment of Dermatovenereology, Chengdu Second People's Hospital, Sichuan, 610011, China.
Zhixuan WuDepartment of Gastroenterology, The Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400010, China.
Song HeDepartment of Gastroenterology, The Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400010, China.
Yongyu ChenDepartment of Gastroenterology, The Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400010, China.ORCID https://orcid.org/0000-0002-2774-374X

Funding

National Natural Science Foundation of China 82100548Natural Science Foundation of Chongqing Municipality CSTB2022NSCQ-MSX0094
6 · The paper itself

Abstract

Approximately 50% of the patients with ulcerative colitis (UC) are primarily nonresponsive to anti-tumor necrosis factor (TNF) therapy or lose their responsiveness over time. The gut microbiota plays an important role in the resistance of UC to anti-TNF therapy; however, the underlying mechanism remains unknown. Here, it is found that the transplantation of gut fecal microbiota from patients with UC alters the diversity of the gut microbiota in dextran sulfate sodium-induced colitis mice and may affect the therapeutic responsiveness of mice to infliximab. Furthermore, the abundances of Romboutsia and Fusobacterium increase in the tissues of patients with UC who do not respond to anti-TNF therapy. Differentially abundant metabolites are mainly enriched in nicotinate and nicotinamide metabolism in NCM460 cells after Fusobacterium nucleatum infection. Mechanistically, F. nucleatum promotes the nicotinamide adenine dinucleotide (NAD

Indexed as

Colitis, UlcerativeGastrointestinal MicrobiomeNADTumor Necrosis Factor-alphaTumor Necrosis Factor InhibitorsAnimalsDisease Models, AnimalFecal Microbiota TransplantationFemaleHumansInfliximabMaleMiceMice, Inbred C57BLInfliximabNADTumor Necrosis Factor-alphaTumor Necrosis Factor Inhibitorsanti‐tumor necrosis factor therapyfusobacterium nucleatummicrobiotaNAD+ salvage pathwayulcerative colitis

Identifiers

PMID39739648
PMCPMC11848563

What Socratic holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.