Evidence mapPaperPMID 39757322Full record

ReviewNature reviews. Endocrinology2025

Bile acid metabolism in type 2 diabetes mellitus.

Marti Cadena Sandoval, Rebecca A Haeusler

Abstract readReview
In one paragraph

Review in Nature reviews. Endocrinology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 27 papers.

0numbers the graph read from it
0cells of the map it votes in
27citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

27 citing papers in PubMed.

  1. Review
  2. A Novel Blend ofFoods (Basel, Switzerland) · 2026
    Article
  3. Article
  4. Article
  5. Article
  6. Gastrodin Ameliorates Type II Diabetes Through the YY1-FXR-Bile Acid Axis.International journal of molecular sciences · 2026
    Article
  7. Article
  8. Review
  9. Review
  10. Review
  11. Review
  12. Review
  13. Review
  14. Review
  15. Review
  16. Review
  17. Article
  18. Muricholic acid mediates puberty initiation via the hypothalamic TGR5 signaling pathway.Proceedings of the National Academy of Sciences of the United States of America · 2025
    Article
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Marti Cadena SandovalNaomi Berrie Diabetes Center, Columbia University Medical Center, New York, NY, USA.ORCID http://orcid.org/0000-0002-8223-0023
Rebecca A HaeuslerNaomi Berrie Diabetes Center, Columbia University Medical Center, New York, NY, USA. rah2130@cumc.columbia.edu.ORCID http://orcid.org/0000-0002-6973-9845

Funding

Translational Biomarker Analytical Core (TBAC)P30DK063608 · NIDDK · COLUMBIA UNIVERSITY HEALTH SCIENCES · 2003 to 2025
$8.5M
The Organoid and Cell Culture CoreP30DK132710 · COLUMBIA UNIVERSITY HEALTH SCIENCES · 2025 to 2025
$1.2M
Insulin regulation of hepatic transportR01DK135298 · COLUMBIA UNIVERSITY HEALTH SCIENCES · 2025 to 2025
$668k
Bile acid composition and insulin sensitivityR01DK115825 · COLUMBIA UNIVERSITY HEALTH SCIENCES · 2025 to 2025
$640k
NIDDK NIH HHS P30 DK063608NIDDK NIH HHS P30 DK132710NIDDK NIH HHS R01 DK115825NIDDK NIH HHS R01 DK135298
6 · The paper itself

Abstract

Type 2 diabetes mellitus is a complex disorder associated with insulin resistance and hyperinsulinaemia that is insufficient to maintain normal glucose metabolism. Changes in insulin signalling and insulin levels are thought to directly explain many of the metabolic abnormalities that occur in diabetes mellitus, such as impaired glucose disposal. However, molecules that are directly affected by abnormal insulin signalling might subsequently go on to cause secondary metabolic effects that contribute to the pathology of type 2 diabetes mellitus. In the past several years, evidence has linked insulin resistance with the concentration, composition and distribution of bile acids. As bile acids are known to regulate glucose metabolism, lipid metabolism and energy balance, these findings suggest that bile acids are potential mediators of metabolic distress in type 2 diabetes mellitus. In this Review, we highlight advances in our understanding of the complex regulation of bile acids during insulin resistance, as well as how bile acids contribute to metabolic control.

Indexed as

Bile Acids and SaltsDiabetes Mellitus, Type 2AnimalsEnergy MetabolismGlucoseHumansInsulinInsulin ResistanceLipid MetabolismSignal TransductionBile Acids and SaltsGlucoseInsulin

Identifiers

PMID39757322
PMCPMC12053743

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.