Evidence map›Paper›PMID 39762120›Full record

ArticleRMD open2025

Combination of clinical factors predicts successful glucocorticoid withdrawal in systemic lupus erythematosus (SLE): results from a multicentre, retrospective cohort study.

Spyridon Katechis, Sofia Pitsigavdaki, Myrto Nikoloudaki, Ettore Silvagni, Argyro Repa, Antonio Marangoni, Irini Flouri, Nestor Avgoustidis, Konstantinos Parperis, Marcello Govoni and 5 more

Abstract readMulticenter Study
In one paragraph

Article in RMD open, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

15 authors.

Spyridon KatechisRheumatology and Clinical Immunology Unit, 4th Department of Internal Medicine, Attikon University Hospital, Joint Rheumatology Program, National and Kapodistrian University of Athens School of Medicine, Athens, Greece.
Sofia PitsigavdakiRheumatology and Clinical Immunology, University of Crete School of Medicine, Heraklion, Greece.
Myrto NikoloudakiRheumatology and Clinical Immunology, University of Crete School of Medicine, Heraklion, Greece.
Ettore SilvagniRheumatology Unit, Department of Medical Sciences, University of Ferrara and Azienda Ospedaliero-Universitaria S.Anna, Ferrara, Italy.
Argyro RepaRheumatology and Clinical Immunology, University of Crete School of Medicine, Heraklion, Greece.
Antonio MarangoniRheumatology Unit, Department of Medical Sciences, University of Ferrara and Azienda Ospedaliero-Universitaria S.Anna, Ferrara, Italy.
Irini FlouriRheumatology and Clinical Immunology, University of Crete School of Medicine, Heraklion, Greece.ORCID 0000-0002-6966-1304
Nestor AvgoustidisRheumatology and Clinical Immunology, University of Crete School of Medicine, Heraklion, Greece.
Konstantinos ParperisDivision of Rheumatology, Department of Medicine, University of Cyprus Medical School, Nicosia, Cyprus.
Marcello GovoniRheumatology Unit, Department of Medical Sciences, University of Ferrara and Azienda Ospedaliero-Universitaria S.Anna, Ferrara, Italy.
Prodromos SidiropoulosRheumatology and Clinical Immunology, University of Crete School of Medicine, Heraklion, Greece.ORCID 0000-0001-9607-0326
Dimitrios T BoumpasRheumatology and Clinical Immunology Unit, 4th Department of Internal Medicine, Attikon University Hospital, Joint Rheumatology Program, National and Kapodistrian University of Athens School of Medicine, Athens, Greece.ORCID 0000-0002-9812-4671
Antonis FanouriakisRheumatology and Clinical Immunology Unit, 4th Department of Internal Medicine, Attikon University Hospital, Joint Rheumatology Program, National and Kapodistrian University of Athens School of Medicine, Athens, Greece.ORCID 0000-0003-2696-031X
George Bertsias *Rheumatology and Clinical Immunology, University of Crete School of Medicine, Heraklion, Greece gbertsias@uoc.gr.ORCID 0000-0001-5299-1406
Alessandra Bortoluzzi *Rheumatology Unit, Department of Medical Sciences, University of Ferrara and Azienda Ospedaliero-Universitaria S.Anna, Ferrara, Italy.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectiveGlucocorticoid (GC) tapering and withdrawal to reduce damage represents a key aspect of the European Alliance of Associations for Rheumatology (EULAR) SLE recommendations. However, optimal strategies for relapse-free GC cessation remain ill-defined. We characterised clinical predictors and their combined effect on flares in patients with SLE who discontinued GC.

methodsRetrospective cohort of 324 patients with active SLE (PGA ≥1.5 and/or SLEDAI-2K ≥6) who received GC as part of treatment intensification (median follow-up 60 months). Survival and generalised linear models estimated SELENA-SLEDAI flare risks and their predictors.

resultsGCs were discontinued in 220 (67.9%) patients with 1-year risks for overall and severe flares of 50% and 25%, respectively (HR: 1.48; 95% CI: 1.12 to 1.96 for overall flares; HR: 1.52; 95% CI: 1.03 to 2.25 for severe flares, compared with non-withdrawers). Flare risk was lowered when GCs were ceased during remission (DORIS) or Lupus Low Disease Activity State (LLDAS; excluding remission) (HR for severe flares: 0.23; 0.12 to 0.43 and 0.30; 0.18 to 0.50, respectively), with each additional month in targets providing further protection. Hydroxychloroquine prevented total (HR: 0.37; 0.26 to 0.53) and severe flares (HR: 0.33; 0.21 to 0.52), while mycophenolate and azathioprine reduced overall flares. Prednisone tapering from 7.5 mg/day to 0 over >6 months improved severe flare-free outcome (HR: 0.57; 0.37 to 0.90). Random survival forests identified DORIS/LLDAS, hydroxychloroquine use and slow GC tapering as top predictors, whose coexistence reduced overall and severe flares by ~25 fold and ~50 fold, respectively. This combination reduced damage (IRR: 0.31; 0.08 to 0.84) without inducing flares (IRR: 0.52; 95% CI: 0.18 to 1.16) compared with GC non-withdrawers.

conclusionLow or absent disease activity, slow tapering and hydroxychloroquine use minimise the risk of flares, facilitating GC discontinuation-a major goal in SLE.

Indexed as

GlucocorticoidsLupus Erythematosus, SystemicAdultDrug TaperingFemaleHumansHydroxychloroquineMaleMiddle AgedRemission InductionRetrospective StudiesSeverity of Illness IndexTreatment OutcomeWithholding TreatmentGlucocorticoidsHydroxychloroquineGlucocorticoidsHydroxychloroquineLupus Erythematosus, Systemic

Identifiers

PMID39762120
PMCPMC11749689

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.