Evidence map›Paper›PMID 39766192›Full record

ReviewBiomolecules2024

Hedgehog Signaling Pathway in Fibrosis and Targeted Therapies.

Yuchen Hu, Linrui Peng, Xinyu Zhuo, Chan Yang, Yuwei Zhang

Abstract readReview
In one paragraph

Review in Biomolecules, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Total Flavonoids fromInternational journal of molecular sciences · 2026
    Article
  2. Review
  3. Curcumin in Fibrosis Treatment: A Review.Drug design, development and therapy · 2026
    Review
  4. Review
  5. Review
  6. Review
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Yuchen HuDepartment of Endocrinology and Metabolism, West China Hospital, Sichuan University, Chengdu 610041, China.
Linrui PengDepartment of Endocrinology and Metabolism, West China Hospital, Sichuan University, Chengdu 610041, China.
Xinyu ZhuoDepartment of Endocrinology and Metabolism, West China Hospital, Sichuan University, Chengdu 610041, China.
Chan YangDivision of Endocrinology and Metabolism, State Key Laboratory of Biotherapy, West China Hospital, Sichuan University and Collaborative Innovation Center of Biotherapy, Chengdu 610041, China.
Yuwei ZhangDepartment of Endocrinology and Metabolism, West China Hospital, Sichuan University, Chengdu 610041, China.ORCID 0000-0003-2281-5000

Funding

National Natural Science Foundation of China 82070660Regional Innovation Cooperation Project of Science and Technology Department of Sichuan Province 2024YFHZ0145Sichuan Provincial Cadre Healthcare Research Program 2023-107
6 · The paper itself

Abstract

Hedgehog (Hh) signaling is a well-established developmental pathway; it is crucial for early embryogenesis, cell differentiation, and damage-driven regeneration. It is being increasingly recognized that dysregulated Hh signaling is also involved in fibrotic diseases, which are characterized by excessive extracellular matrix deposition that compromises tissue architecture and function. As in-depth insights into the mechanisms of Hh signaling are obtained, its complex involvement in fibrosis is gradually being illuminated. Notably, some Hh-targeted inhibitors are currently under exploration in preclinical and clinical trials as a means to prevent fibrosis progression. In this review, we provide a concise overview of the biological mechanisms involved in Hh signaling. We summarize the latest advances in our understanding of the roles of Hh signaling in fibrogenesis across the liver, kidneys, airways, and lungs, as well as other tissues and organs, with an emphasis on both the shared features and, more critically, the distinct functional variations observed across these tissues and organs. We thus highlight the context dependence of Hh signaling, as well as discuss the current status and the challenges of Hh-targeted therapies for fibrosis.

Indexed as

FibrosisHedgehog ProteinsSignal TransductionAnimalsHumansMolecular Targeted TherapyHedgehog Proteinsfibrosishedgehog signaling pathwaytargeted therapies

Identifiers

PMID39766192
PMCPMC11727624

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.