Evidence map›Paper›PMID 39769450›Full record

ReviewInternational journal of molecular sciences2024

Therapeutic Significance of NLRP3 Inflammasome in Cancer: Friend or Foe?

Aliea M Jalali, Kenyon J Mitchell, Christian Pompoco, Sudeep Poludasu, Sabrina Tran, Kota V Ramana

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2024. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed.

  1. Review
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  10. Programmed cell death in triple-negative breast cancer.Cellular & molecular biology letters · 2025
    Review
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  12. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Aliea M JalaliDepartment of Biomedical Sciences, Noorda College of Osteopathic Medicine, Provo, UT 84606, USA.ORCID 0000-0002-8276-4092
Kenyon J MitchellDepartment of Biomedical Sciences, Noorda College of Osteopathic Medicine, Provo, UT 84606, USA.
Christian PompocoDepartment of Biomedical Sciences, Noorda College of Osteopathic Medicine, Provo, UT 84606, USA.
Sudeep PoludasuDepartment of Biomedical Sciences, Noorda College of Osteopathic Medicine, Provo, UT 84606, USA.
Sabrina TranDepartment of Biomedical Sciences, Noorda College of Osteopathic Medicine, Provo, UT 84606, USA.
Kota V RamanaDepartment of Biomedical Sciences, Noorda College of Osteopathic Medicine, Provo, UT 84606, USA.ORCID 0000-0001-6502-7800

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Besides various infectious and inflammatory complications, recent studies also indicated the significance of NLRP3 inflammasome in cancer progression and therapy. NLRP3-mediated immune response and pyroptosis could be helpful or harmful in the progression of cancer, and also depend on the nature of the tumor microenvironment. The activation of NLRP3 inflammasome could increase immune surveillance and the efficacy of immunotherapy. It can also lead to the removal of tumor cells by the recruitment of phagocytic macrophages, T-lymphocytes, and other immune cells to the tumor site. On the other hand, NLRP3 activation can also be harmful, as chronic inflammation driven by NLRP3 supports tumor progression by creating an environment that facilitates cancer cell proliferation, migration, invasion, and metastasis. The release of pro-inflammatory cytokines such as IL-1β and IL-18 can promote tumor growth and angiogenesis, while sustained inflammation may lead to immune suppression, hindering effective anti-tumor responses. In this review article, we discuss the role of NLRP3 inflammasome-mediated inflammatory response in the pathophysiology of various cancer types; understanding this role is essential for the development of innovative therapeutic strategies for cancer growth and spread.

Indexed as

InflammasomesNeoplasmsNLR Family, Pyrin Domain-Containing 3 ProteinAnimalsHumansImmunotherapyInflammationTumor MicroenvironmentInflammasomesNLR Family, Pyrin Domain-Containing 3 ProteinNLRP3 protein, humancancercaspase-1IL-1binflammasomeinnate immunityNLRP3

Identifiers

PMID39769450
PMCPMC11728390

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.