Evidence mapPaperPMID 39827271Full record

ArticleNature communications2025

α-Synuclein fibrils enhance HIV-1 infection of human T cells, macrophages and microglia.

Lia-Raluca Olari, Sichen Liu, Franziska Arnold, Julia Kühlwein, Marta Gil Miró, Ajeet Rijal Updahaya, Christina Stürzel, Dietmar Rudolf Thal, Paul Walther, Konstantin M J Sparrer and 3 more

Abstract read
In one paragraph

Article in Nature communications, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Lia-Raluca Olari *Institute of Molecular Virology, Ulm University Medical Center, 89081, Ulm, Germany.ORCID http://orcid.org/0000-0001-7793-4408
Sichen Liu *Institute of Molecular Virology, Ulm University Medical Center, 89081, Ulm, Germany.ORCID http://orcid.org/0009-0006-1023-9076
Franziska ArnoldInstitute of Molecular Virology, Ulm University Medical Center, 89081, Ulm, Germany.
Julia KühlweinDepartment of Neurology, Ulm University, 89081, Ulm, Germany.
Marta Gil MiróInstitute of Molecular Virology, Ulm University Medical Center, 89081, Ulm, Germany.
Ajeet Rijal UpdahayaLaboratory of Neuropathology, Institute of Pathology, Center for Clinical Research at the University of Ulm, 89081, Ulm, Germany.
Christina StürzelInstitute of Molecular Virology, Ulm University Medical Center, 89081, Ulm, Germany.
Dietmar Rudolf ThalLaboratory of Neuropathology, Institute of Pathology, Center for Clinical Research at the University of Ulm, 89081, Ulm, Germany.ORCID http://orcid.org/0000-0002-1036-1075
Paul WaltherCentral Facility for Electron Microscopy, Ulm University, 89081, Ulm, Germany.
Konstantin M J SparrerInstitute of Molecular Virology, Ulm University Medical Center, 89081, Ulm, Germany.ORCID http://orcid.org/0000-0002-8682-1779
Karin M DanzerDepartment of Neurology, Ulm University, 89081, Ulm, Germany.ORCID http://orcid.org/0000-0003-0133-7046
Jan MünchInstitute of Molecular Virology, Ulm University Medical Center, 89081, Ulm, Germany.ORCID http://orcid.org/0000-0001-7316-7141
Frank KirchhoffInstitute of Molecular Virology, Ulm University Medical Center, 89081, Ulm, Germany. frank.kirchhoff@uni-ulm.de.ORCID http://orcid.org/0000-0002-7052-2360

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

HIV-associated neurocognitive disorders (HAND) and viral reservoirs in the brain remain a significant challenge. Despite their importance, the mechanisms allowing HIV-1 entry and replication in the central nervous system (CNS) are poorly understood. Here, we show that α-synuclein and (to a lesser extent) Aβ fibrils associated with neurological diseases enhance HIV-1 entry and replication in human T cells, macrophages, and microglia. Additionally, an HIV-1 Env-derived amyloidogenic peptide accelerated amyloid formation by α-synuclein and Aβ peptides. Mechanistic studies show that α-synuclein and Aβ fibrils interact with HIV-1 particles and promote virion attachment and fusion with target cells. Despite an overall negative surface charge, these fibrils facilitate interactions between viral and cellular membranes. The enhancing effects of human brain extracts on HIV-1 infection correlated with their binding to Thioflavin T, a dye commonly used to stain amyloids. Our results suggest a detrimental interplay between HIV-1 and brain amyloids that may contribute to the development of neurodegenerative diseases.

Indexed as

alpha-SynucleinAmyloidHIV-1HIV InfectionsMacrophagesMicrogliaT-LymphocytesAmyloid beta-PeptidesBrainHumansVirus InternalizationVirus Replicationalpha-SynucleinAmyloidAmyloid beta-Peptides

Identifiers

PMID39827271
PMCPMC11742913

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.