Evidence map›Paper›PMID 39829685›Full record

ArticleClinical diabetes : a publication of the American Diabetes Association2025

Metformin Improves Glycemic Control and Postprandial Metabolism and Enhances Postprandial Glucagon-Like Peptide 1 Secretion in Patients With Type 2 Diabetes and Heart Failure: A Randomized, Double-Blind, Placebo-Controlled Trial.

Vojtěch Melenovský, Eva Hošková, Kateřina Velebová, Jiří Veleba, Barry A Borlaug, Jan Benes, Ondřej Kuda, Tomáš Čajka, Markéta Segeťová, Lenka Thieme and 6 more

Abstract read
In one paragraph

Article in Clinical diabetes : a publication of the American Diabetes Association, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors.

Vojtěch MelenovskýInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Eva HoškováInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Kateřina VelebováInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Jiří VelebaInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Barry A BorlaugDepartment of Cardiovascular Medicine, Mayo Clinic, Rochester, MN.
Jan BenesInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Ondřej KudaInstitute of Physiology of the Czech Academy of Sciences, Prague, Czech Republic.
Tomáš ČajkaInstitute of Physiology of the Czech Academy of Sciences, Prague, Czech Republic.
Markéta SegeťováInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Lenka ThiemeInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Jan KopeckýInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Jan KopeckýInstitute of Physiology of the Czech Academy of Sciences, Prague, Czech Republic.
Terezie PelikánováInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Martin HaluzíkInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.
Martin HillInstitute of Endocrinology, Prague, Czech Republic.
Hana KahleováInstitute for Clinical and Experimental Medicine, Prague, Czech Republic.ORCID https://orcid.org/0000-0003-0491-3993

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

This randomized trial tested the effect of metformin on glycemic control and cardiac function in patients with heart failure (HF) and type 2 diabetes while evaluating intestinal effects on selected gut microbiome products reflected by trimethylamine-N-oxide (TMAO) and gut-derived incretins. Metformin treatment improved glycemic control and postprandial metabolism and enhanced postprandial glucagon-like peptide 1 (GLP-1) secretion but did not influence cardiac function or the TMAO levels. Metabolic effects of metformin in HF may be mediated by an improvement in intestinal endocrine function and enhanced secretion of the gut-derived incretin GLP-1.

Identifiers

PMID39829685
PMCPMC11739370

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.