Evidence mapPaperPMID 39833533Full record

ReviewNature reviews. Cancer2025

Metabolic interplays between the tumour and the host shape the tumour macroenvironment.

Patricia Altea-Manzano, Amanda Decker-Farrell, Tobias Janowitz, Ayelet Erez

Abstract readReview
In one paragraph

Review in Nature reviews. Cancer, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 55 papers.

0numbers the graph read from it
0cells of the map it votes in
55citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

55 citing papers in PubMed.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Patricia Altea-Manzano *Andalusian Molecular Biology and Regenerative Medicine Centre-CSIC, Sevilla, Spain. patricia.altea@cabimer.es.ORCID http://orcid.org/0000-0002-7075-2051
Amanda Decker-Farrell *Cold Spring Harbor Laboratory, Cold Spring Harbor, NY, USA.ORCID http://orcid.org/0000-0001-9969-7909
Tobias JanowitzCold Spring Harbor Laboratory, Cold Spring Harbor, NY, USA.ORCID http://orcid.org/0000-0002-7820-3727
Ayelet ErezWeizmann Institute of Science, Rehovot, Israel. ayelet.erez@weizmann.ac.il.ORCID http://orcid.org/0000-0002-9696-0393

Funding

Single-Cell Biology Shared ResourceP30CA045508 · COLD SPRING HARBOR LABORATORY · 1987 to 2025
$27.2M
CANCAN - COLDSPRINGOT2CA278690 · NCI · COLD SPRING HARBOR LABORATORY · 2022 to 2025
$1.2M
Mechanism by which ketogenic diet uncouples tumor growth and overall survivalR37CA286477 · COLD SPRING HARBOR LABORATORY · 2025 to 2025
$533k
NCI NIH HHS OT2 CA278690NCI NIH HHS P30 CA045508NCI NIH HHS R37 CA286477
6 · The paper itself

Abstract

Metabolic reprogramming of cancer cells and the tumour microenvironment are pivotal characteristics of cancers, and studying these processes offer insights and avenues for cancer diagnostics and therapeutics. Recent advancements have underscored the impact of host systemic features, termed macroenvironment, on facilitating cancer progression. During tumorigenesis, these inherent features of the host, such as germline genetics, immune profile and the metabolic status, influence how the body responds to cancer. In parallel, as cancer grows, it induces systemic effects beyond the primary tumour site and affects the macroenvironment, for example, through inflammation, the metabolic end-stage syndrome of cachexia, and metabolic dysregulation. Therefore, understanding the intricate metabolic interplay between the tumour and the host is a growing frontier in advancing cancer diagnosis and therapy. In this Review, we explore the specific contribution of the metabolic fitness of the host to cancer initiation, progression and response to therapy. We then delineate the complex metabolic crosstalk between the tumour, the microenvironment and the host, which promotes disease progression to metastasis and cachexia. The metabolic relationships among the host, cancer pathogenesis and the consequent responsive systemic manifestations during cancer progression provide new perspectives for mechanistic cancer therapy and improved management of patients with cancer.

Indexed as

NeoplasmsTumor MicroenvironmentAnimalsCachexiaDisease ProgressionHumans

Identifiers

PMID39833533
PMCPMC12140436

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.