Article in American journal of physiology. Heart and circulatory physiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Nicole TaubeDepartment of Environmental Health and Engineering, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland, United States.
Morgan SteinerDepartment of Environmental Health and Engineering, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland, United States.ORCID 0009-0008-3974-9154
Obialunanma V Ebenebe-KasondeDepartment of Environmental Health and Engineering, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland, United States.ORCID 0000-0001-5238-928X
Raihan KabirDepartment of Environmental Health and Engineering, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland, United States.ORCID 0000-0001-5863-5760
Haley Garbus-GrantDepartment of Environmental Health and Engineering, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland, United States.
Sarah-Marie Alam El DinDepartment of Environmental Health and Engineering, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland, United States.
Emily IllingworthDepartment of Environmental Health and Engineering, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland, United States.
Nadan WangCardiology Division, Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, Maryland, United States.ORCID 0000-0002-1682-080X
Brian L LinCardiology Division, Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, Maryland, United States.ORCID 0000-0003-0800-7103
Mark J KohrDepartment of Environmental Health and Engineering, Johns Hopkins Bloomberg School of Public Health, Baltimore, Maryland, United States.ORCID 0000-0002-6034-5962
Funding
Traning Program in Environmental Health SciencesT32ES007141 · NIEHS · JOHNS HOPKINS UNIVERSITY · PI Marsha Wills-Karp · 1985 to 2026
$22.2M
S-nitrosation in cell survival and cell deathR01HL136496 · NHLBI · JOHNS HOPKINS UNIVERSITY · PI KOHR, MARK JEFFREY · 2018 to 2022
$2.0M
TRPC6 inhibition therapy to rescue cardiac muscle dysfunction in muscular dystrophyR00HL155840 · NHLBI · MEDICAL COLLEGE OF WISCONSIN · PI Brian Leei Lin · 2024 to 2026
$747k
Role of formaldehyde, formate and one-carbon metabolism in the female heartR21HL157800 · NHLBI · JOHNS HOPKINS UNIVERSITY · PI KOHR, MARK JEFFREY · 2021 to 2022
$450k
Sex Differences in Endogenous Formaldehyde Production and One-Carbon Metabolism: Potential Links to CardioprotectionF31HL165820 · NHLBI · JOHNS HOPKINS UNIVERSITY · PI GARBUS, HALEY CAITLIN · 2022 to 2023
$74k
HHS | National Institutes of Health (NIH) ES007141HHS | National Institutes of Health (NIH) HL136496HHS | National Institutes of Health (NIH) HL157800HHS | National Institutes of Health (NIH) HL165820NHLBI NIH HHS F31 HL165820NHLBI NIH HHS R00 HL155840NHLBI NIH HHS R01 HL136496NHLBI NIH HHS R21 HL157800NIEHS NIH HHS T32 ES007141
6 · The paper itself
Abstract
Cardiovascular disease is the leading cause of mortality in the United States. Studies suggest a role for environmental exposures in the etiology of cardiovascular disease, including exposure to arsenic through drinking water. Arsenic exposure during pregnancy has been shown to have effects on offspring, but few studies have examined impacts on maternal cardiovascular health. Although our prior work documented the detrimental effect of arsenic on the maternal heart during pregnancy, our current study examines the effect of gestational arsenic exposure on the maternal heart postpartum. Timed-pregnant wild-type (C57BL/6J) mice were exposed to 0, 100, or 1,000 µg/L sodium arsenite (NaAsO
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.
Gestational arsenite exposure alters maternal postpartum heart size and induces Ca · full record | Socratic