Evidence mapPaperPMID 39950467Full record

ReviewProtein and peptide letters2025

A Review on the Potential Role of Humanin Peptide and its Analogs in the Regulation of Autophagy Pathways for Therapeutic Application in Metabolic Disorders.

Hira Moin, Rizwan Ashraf, Batool Butt, Imtiaz Mustafa, Mamoona Shafiq, Syed Ali Raza Shah

Abstract readReview
PubMed Publisher
In one paragraph

Review in Protein and peptide letters, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Hira MoinInstitute of Molecular Biology and Biotechnology, The University of Lahore, Lahore, Pakistan.ORCID 0000-0002-8793-3844
Rizwan AshrafNUST School of Health Sciences, National University of Sciences and Technology, Islamabad, 44000, Pakistan.
Batool ButtDepartment of Nephrology, Foundation University School of Health Sciences, Fauji Foundation Hospitals, Islamabad, Pakistan.
Imtiaz MustafaInstitute of Molecular Biology and Biotechnology, The University of Lahore, Lahore, Pakistan.
Mamoona ShafiqInstitute of Molecular Biology and Biotechnology, The University of Lahore, Lahore, Pakistan.
Syed Ali Raza ShahInstitute of Molecular Biology and Biotechnology, The University of Lahore, Lahore, Pakistan.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Autophagy is a self-eating cellular process in which the cell breaks down worn-out organelles, damaged/defective proteins, and toxins. Impaired autophagy is a significant factor in the development of various metabolic disorders, along with oxidative stress, inflammation, mitochondrial and endoplasmic reticulum dysfunction. These disorders pose a significant health and economic burden on the global human population, owing to their steadily rising prevalence. Therefore, modulating the expression of proteins involved in the autophagy-related pathways can be a promising avenue for curbing the development and progression of these disorders. Humanin (HN) is a 24-amino acid mitochondrial-derived peptide. It possesses anti-oxidant, anti-inflammatory, and pro-apoptotic properties. The analogs of HN can be generated by replacing specific amino acids in the polypeptide chain, thereby functionally modifying the peptide. Among these, humanin- glycine (HNG) is the most widely studied analog in both

Indexed as

AutophagyIntracellular Signaling Peptides and ProteinsMetabolic SyndromeAnimalsCell SurvivalHumansObesityhumaninIntracellular Signaling Peptides and ProteinsAutophagydiabetesHNGhumaninmetabolic diseasespolypeptide chain.

Identifiers

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.