Evidence map›Paper›PMID 40068814›Full record

ArticleOpen biology2025

Evolution of resistance and disease tolerance mechanisms to oral bacterial infection in

Tânia F Paulo, Priscilla A Akyaw, Tiago Paixão, Élio Sucena

Abstract read
In one paragraph

Article in Open biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

  1. When Does Stress Build Strength? Linking Stress Physiology to Resilience in the Age of Rapid Environmental Change.BioEssays : news and reviews in molecular, cellular and developmental biology · 2026
    Review
  2. Sex differences inProceedings of the National Academy of Sciences of the United States of America · 2026
    Article
  3. Article
  4. Article
  5. The "yeast-drop" treatment protocol in theFrontiers in tuberculosis · 2026
    Article
  6. Standard ingredient ofMicrobiology spectrum · 2025
    Article
  7. Article
  8. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Tânia F PauloInstituto Gulbenkian de Ciência, Oeiras, Portugal.ORCID 0000-0002-8815-2636
Priscilla A AkyawInstituto Gulbenkian de Ciência, Oeiras, Portugal.ORCID 0000-0003-0517-6358
Tiago PaixãoInstituto Gulbenkian de Ciência, Oeiras, Portugal.ORCID 0000-0003-2361-3953
Élio SucenaInstituto Gulbenkian de Ciência, Oeiras, Portugal.ORCID 0000-0001-8810-870X

Funding

CONGENTOFCT-Fundação para a Ciência e a Tecnologia (Portugal)Instituto Gulbenkian de Ciência/Fundação Calouste Gulbenkian
6 · The paper itself

Abstract

Pathogens exert strong selection on hosts that evolve and deploy different defensive strategies, namely minimizing pathogen exposure (avoidance), directly promoting pathogen elimination (resistance) and/or managing the deleterious effects of illness (disease tolerance). However, how the host response partitions across these processes has not been directly tested in a single host-pathogen system, let alone in the context of known adaptive trajectories resulting from experimental evolution. Here, we compare a

Indexed as

Biological EvolutionDisease ResistanceDrosophila melanogasterHost-Pathogen InteractionsPseudomonasPseudomonas InfectionsAnimalsdisease toleranceDrosophilaexperimental evolutioninnate immunityresistance

Identifiers

PMID40068814
PMCPMC11896704

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.