Evidence map›Paper›PMID 40122162›Full record

ReviewThe Canadian journal of cardiology2025

Insights Into the Role of Obesity in Heart Failure With Preserved Ejection Fraction: Pathophysiology and Management.

Brandon Hathorn, Mark J Haykowsky, Jaime Almandoz, Ambarish Pandey, Satyam Sarma, Christopher M Hearon, Tony G Babb, Bryce N Balmain, Qi Fu, Vlad G Zaha and 2 more

Abstract readReview
In one paragraph

Review in The Canadian journal of cardiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Review
  2. Review
  3. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Brandon HathornApplied Physiology and Advanced Imaging Laboratory, University of Texas at Arlington, Arlington, Texas, USA.
Mark J HaykowskyCollege of Health Sciences, Faculty of Nursing, University of Alberta, Edmonton, Alberta, Canada.
Jaime AlmandozDivision of Endocrinology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Ambarish PandeyDivision of Cardiology, Department of Internal Medicine, UT Southwestern Medical Center, Dallas, Texas, USA.
Satyam SarmaDivision of Cardiology, Department of Internal Medicine, UT Southwestern Medical Center, Dallas, Texas, USA; Institute for Exercise and Environmental Medicine, Texas Health Presbyterian Dallas, Dallas, Texas, USA.
Christopher M HearonInstitute for Exercise and Environmental Medicine, Texas Health Presbyterian Dallas, Dallas, Texas, USA.
Tony G BabbInstitute for Exercise and Environmental Medicine, Texas Health Presbyterian Dallas, Dallas, Texas, USA; Division of Pulmonary and Critical Care Medicine, Department of Internal Medicine, UT Southwestern Medical Center, Dallas, Texas, USA.
Bryce N BalmainInstitute for Exercise and Environmental Medicine, Texas Health Presbyterian Dallas, Dallas, Texas, USA; Division of Pulmonary and Critical Care Medicine, Department of Internal Medicine, UT Southwestern Medical Center, Dallas, Texas, USA.
Qi FuDivision of Cardiology, Department of Internal Medicine, UT Southwestern Medical Center, Dallas, Texas, USA; Institute for Exercise and Environmental Medicine, Texas Health Presbyterian Dallas, Dallas, Texas, USA.
Vlad G ZahaDivision of Cardiology, Department of Internal Medicine, UT Southwestern Medical Center, Dallas, Texas, USA; Clinical Imaging Research Center, University of Texas at Arlington, Arlington, Texas, USA.
Benjamin D LevineDivision of Cardiology, Department of Internal Medicine, UT Southwestern Medical Center, Dallas, Texas, USA; Institute for Exercise and Environmental Medicine, Texas Health Presbyterian Dallas, Dallas, Texas, USA.
Michael D NelsonApplied Physiology and Advanced Imaging Laboratory, University of Texas at Arlington, Arlington, Texas, USA; Division of Cardiology, Department of Internal Medicine, UT Southwestern Medical Center, Dallas, Texas, USA; Clinical Imaging Research Center, University of Texas at Arlington, Arlington, Texas, USA. Electronic address: michael.nelson3@uta.edu.

Funding

Targeting skeletal muscle to improve exercise capacity in heart failure with preserved ejection fraction.P01HL137630 · NHLBI · UT SOUTHWESTERN MEDICAL CENTER · PI BABB, TONY G · 2019 to 2023
$11.4M
UT Southwestern NORCP30DK127984 · NIDDK · UT SOUTHWESTERN MEDICAL CENTER · PI Jeffrey M Zigman · 2022 to 2026
$7.4M
Polypill strategy for the evidence-based management of heart failure with reduced ejection fraction in an underserved patient populationR01MD017529 · NIMHD · UT SOUTHWESTERN MEDICAL CENTER · PI PANDEY, AMBARISH, WANG, THOMAS J. · 2022 to 2025
$2.9M
Cardiac biomarkers, intensive blood pressure treatment and risk of adverse cardiovascular outcomes in type 2 diabetes, a secondary analysis of the ACCORD BPR21HL169708 · NHLBI · UT SOUTHWESTERN MEDICAL CENTER · PI PANDEY, AMBARISH · 2023 to 2024
$261k
NHLBI NIH HHS P01 HL137630NHLBI NIH HHS R21 HL169708NIDDK NIH HHS P30 DK127984NIMHD NIH HHS R01 MD017529
6 · The paper itself

Abstract

Heart failure (HF) is a significant global health issue, categorized by left ventricular ejection fraction, being either reduced (HFrEF < 0.40) or preserved (HFpEF > 0.50), or in the middle of this range. Although the overall incidence of HF remains stable, HFpEF cases are increasing, representing about 50% of all HF cases. Outcomes for HFpEF are similar to those for HFrEF, leading to substantial health-care resource use. Despite extensive research over the past 2 decades, the prognosis and mortality rates for HFpEF remain high. A key feature of HFpEF is exercise intolerance, characterized by severe exertional dyspnea and fatigue, which significantly impacts quality of life. The underlying mechanisms of exercise intolerance are not fully understood due to the complex pathophysiology and multisystem involvement. Obesity is a common comorbidity in HFpEF, especially in North America, leading to worsening symptoms, hemodynamics, and mortality rates. Increased adiposity leads to inflammation, hypertension, dyslipidemia, and insulin resistance, and impairing cardiac, vascular, pulmonary, and skeletal muscle function. Therefore, managing obesity is crucial in treating HFpEF. In this review we explore the pathophysiologic mechanisms of HFpEF, emphasizing obesity's role, and we discuss current management strategies while identifying areas needing further research.

Indexed as

Disease ManagementHeart FailureObesityStroke VolumeExercise ToleranceHumansPrognosisQuality of Life

Identifiers

PMID40122162
PMCPMC13160563

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.