Evidence mapPaperPMID 40142155Full record

ReviewMolecules (Basel, Switzerland)2025

Targeting the CXCR4/CXCL12 Axis in Cancer Therapy: Analysis of Recent Advances in the Development of Potential Anticancer Agents.

Gerardina Smaldone, Francesca Di Matteo, Roberta Castelluccio, Valeria Napolitano, Maria Rosaria Miranda, Michele Manfra, Pietro Campiglia, Vincenzo Vestuto

Abstract readReview
In one paragraph

Review in Molecules (Basel, Switzerland), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed.

  1. Article
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  15. Could circulating tumor cells explain why breast conservation improves survival despite higher locoregional recurrence?Translational breast cancer research : a journal focusing on translational research in breast cancer · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Gerardina SmaldoneDepartment of Pharmacy, University of Salerno, Via G. Paolo II, 84084 Fisciano, Italy.ORCID 0000-0003-1380-9717
Francesca Di MatteoDepartment of Pharmacy, University of Salerno, Via G. Paolo II, 84084 Fisciano, Italy.ORCID 0000-0002-9907-6472
Roberta CastelluccioDepartment of Pharmacy, University of Salerno, Via G. Paolo II, 84084 Fisciano, Italy.
Valeria NapolitanoDepartment of Pharmacy, University of Salerno, Via G. Paolo II, 84084 Fisciano, Italy.ORCID 0000-0003-2328-2824
Maria Rosaria MirandaPhD Program in Drug Discovery and Development, University of Salerno, 84084 Fisciano, Italy.ORCID 0009-0005-8296-1888
Michele ManfraDepartment of Health Science, University of Basilicata, Viale dell'Ateneo Lucano 10, 85100 Potenza, Italy.ORCID 0000-0002-3320-972X
Pietro CampigliaDepartment of Pharmacy, University of Salerno, Via G. Paolo II, 84084 Fisciano, Italy.ORCID 0000-0002-1069-2181
Vincenzo VestutoDepartment of Pharmacy, University of Salerno, Via G. Paolo II, 84084 Fisciano, Italy.ORCID 0000-0002-3603-5526

Funding

MIUR B53C22006090001
6 · The paper itself

Abstract

Cancer, a leading cause of premature death, arises from genetic and epigenetic mutations that transform normal cells into tumor cells, enabling them to proliferate, evade cell death, and stimulate angiogenesis. Recent evidence indicates that chemokines are essential in tumor development, activating receptors that promote proliferation, invasion, and metastasis. The CXCR4/CXCL12 signaling pathway is gaining attention as a promising target for cancer therapy. CXCR4, a chemokine receptor, is often overexpressed in various types of cancer, including kidney, lung, brain, prostate, breast, pancreas, ovarian, and melanomas. When it binds to its endogenous ligand, CXCL12, it promotes cell survival, proliferation, and migration, crucial mechanisms for the retention of hematopoietic stem cells in the bone marrow and the movement of lymphocytes. The extensive expression of CXCR4 in cancer, coupled with the constant presence of CXCL12 in various organs, drives the activation of this axis, which in turn facilitates angiogenesis, tumor progression, and metastasis. Given the detrimental role of the CXCR4/CXCL12 axis, the search for drugs acting selectively against this protein represents an open challenge. This review aims to summarize the recent advancements in the design and development of CXCR4 antagonists as potential anticancer agents.

Indexed as

Antineoplastic AgentsChemokine CXCL12NeoplasmsReceptors, CXCR4AnimalsHumansMolecular Targeted TherapySignal TransductionAntineoplastic AgentsChemokine CXCL12CXCL12 protein, humanCXCR4 protein, humanReceptors, CXCR4antitumor drugscancerCXCR4/CXCL12 axissmall molecules

Identifiers

PMID40142155
PMCPMC11945090

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.