Evidence map›Paper›PMID 40208704›Full record

ArticleDiabetes care2025

Effect of Marked Weight Loss on Adipose Tissue Biology in People With Obesity and Type 2 Diabetes.

Dmitri Samovski, Gordon I Smith, Hector Palacios, Terri Pietka, Anja Fuchs, Gary J Patti, Allah Nawaz, C Ronald Kahn, Samuel Klein

Registry-linked trialAbstract read
In one paragraph

Article in Diabetes care, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT02207777 (Weight Loss-Independent Metabolic Effects of Roux-En-Y Gastric Bypass in Diabetes), which is not on this map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02207777 nacompletednot on this map

Weight Loss-Independent Metabolic Effects of Roux-En-Y Gastric Bypass in Diabetes

TypeinterventionalSponsorWashington University School of MedicineRan2014 to 2019Enrolled65ConditionsObesity, Type 2 DiabetesArmsRoux-en-Y gastric bypass surgery, Low-calorie diet
3 · Its place in the literature

Who cites it

10 citing papers in PubMed.

  1. Review
  2. Review
  3. Article
  4. Review
  5. Article
  6. SerpinA3k Deficiency Ameliorates Experimental type 2 Diabetes.Cellular and molecular life sciences : CMLS · 2025
    Article
  7. Article
  8. Article
  9. Article
  10. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

9 authors.

Dmitri SamovskiCenter for Human Nutrition, Washington University School of Medicine, St. Louis, MO.ORCID 0000-0002-6860-6688
Gordon I SmithCenter for Human Nutrition, Washington University School of Medicine, St. Louis, MO.ORCID 0000-0003-0063-046X
Hector PalaciosCenter for Human Nutrition, Washington University School of Medicine, St. Louis, MO.
Terri PietkaCenter for Human Nutrition, Washington University School of Medicine, St. Louis, MO.
Anja FuchsCenter for Human Nutrition, Washington University School of Medicine, St. Louis, MO.
Gary J PattiDepartment of Chemistry, Washington University School of Medicine, St. Louis, MO.
Allah NawazSection of Integrative Physiology and Metabolism, Joslin Diabetes Center, Department of Medicine, Harvard Medical School, Boston, MA.
C Ronald KahnSection of Integrative Physiology and Metabolism, Joslin Diabetes Center, Department of Medicine, Harvard Medical School, Boston, MA.ORCID 0000-0002-7583-9228
Samuel KleinCenter for Human Nutrition, Washington University School of Medicine, St. Louis, MO.ORCID 0000-0001-7127-1156

Funding

WU INSTITUTE OF CLINICAL AND TRANSLATIONAL SCIENCESUL1TR002345 · NCATS · WASHINGTON UNIVERSITY · PI William G. Powderly · 2017 to 2026
$97.8M
SPECIAL ASSAY COREP30DK036836 · NIDDK · JOSLIN DIABETES CENTER · PI JEAN E. SCHAFFER · 1986 to 2026
$50.5M
Washington University Nutrition Obesity Research CenterP30DK056341 · NIDDK · WASHINGTON UNIVERSITY · PI Jonathan R Brestoff · 1999 to 2026
$30.2M
WU P&FP30DK020579 · NIDDK · WASHINGTON UNIVERSITY · PI Clay F. Semenkovich · 2013 to 2026
$27.1M
Washington University Center for Diabetes Translation Research P30DK092950 · NIDDK · WASHINGTON UNIVERSITY · PI Ross C Brownson, Debra Haire-Joshu · 2011 to 2026
$11.7M
Developmental genes, miRNAs and adipose tissueR01DK082659 · NIDDK · JOSLIN DIABETES CENTER · PI C RONALD KAHN · 2009 to 2026
$10.7M
WUSTL Transdisciplinary Pre- and Postdoctoral Training Program in Obesity and Cardiovascular DiseaseT32HL130357 · NHLBI · WASHINGTON UNIVERSITY · PI WILFLEY, DENISE ELLA · 2016 to 2025
$4.3M
The Insulin Receptor and Its Signaling MechanismsR01DK031036 · NIDDK · JOSLIN DIABETES CENTER · PI C RONALD KAHN · 1986 to 2026
$4.2M
Exosomes and insulin action in metabolically healthy and unhealthy obesityR01DK137206 · NIDDK · WASHINGTON UNIVERSITY · PI Samuel Klein, Dmitri Samovski · 2023 to 2026
$3.0M
Alterations in Post-Receptor Insulin Signaling in Diabetes and Insulin ResistanceR01DK128429 · NIDDK · JOSLIN DIABETES CENTER · PI KAHN, C RONALD · 2021 to 2025
$2.9M
WEIGHT LOSS-INDEPENDENT METABOLIC EFFECTS OF ROUX-EN-Y GASTRIC BYPASS IN DIABETESR01DK101578 · NIDDK · WASHINGTON UNIVERSITY · PI KLEIN, SAMUEL · 2014 to 2018
$2.5M
Division of Diabetes, Endocrinology, and Metabolic Diseases P30DK20579Division of Diabetes, Endocrinology, and Metabolic Diseases P30DK56341Division of Diabetes, Endocrinology, and Metabolic Diseases R01DK031036Division of Diabetes, Endocrinology, and Metabolic Diseases R01DK082659Division of Diabetes, Endocrinology, and Metabolic Diseases R01DK101578Division of Diabetes, Endocrinology, and Metabolic Diseases R01DK128429Division of Diabetes, Endocrinology, and Metabolic Diseases R01DK137206Foundation for Barnes-Jewish HospitalNCATS NIH HHS UL1 TR002345NCATS NIH HHS UL1TR002345NHLBI NIH HHS T32 HL130357NIDDK NIH HHS P30 DK020579NIDDK NIH HHS P30 DK036836NIDDK NIH HHS P30 DK056341NIDDK NIH HHS P30 DK092950NIDDK NIH HHS R01 DK031036NIDDK NIH HHS R01 DK082659NIDDK NIH HHS R01 DK101578NIDDK NIH HHS R01 DK128429NIDDK NIH HHS R01 DK137206
6 · The paper itself

Abstract

objectiveWeight loss improves insulin sensitivity in people with obesity and type 2 diabetes. However, the mechanisms responsible for this effect are unclear. We hypothesized that alterations in adipose tissue biology and adipose tissue-related factors in plasma are involved in mediating the systemic metabolic benefits of weight loss. RESEARCH DESIGN AND

methodsWe evaluated blood and adipose tissue samples obtained from 10 adults with obesity and type 2 diabetes before and after marked (16-20%) weight loss and >50% increase in whole-body insulin sensitivity, assessed by using the hyperinsulinemic-euglycemic clamp procedure.

resultsWeight loss 1) decreased adipose tissue expression of genes related to extracellular matrix remodeling; 2) decreased adipose tissue expression of SERPINE 1, which encodes plasminogen activator inhibitor 1 (PAI-1); 3) did not decrease adipose tissue immune cell content or expression of genes involved in inflammation; 4) decreased adipose tissue ceramide content; 5) decreased plasma PAI-1 and leptin concentrations and increased plasma high-molecular weight (HMW) adiponectin; and 6) decreased plasma small extracellular vesicle (sEV) concentration and the sEV content of microRNAs proposed to inhibit insulin action, and completely reversed the inhibitory effect of plasma sEVs on insulin signaling in myotubes.

conclusionsThese findings suggest that weight loss increases insulin sensitivity in people with obesity and type 2 diabetes by modifying adipose tissue biology, with concomitant alterations in circulating PAI-1, leptin, HMW adiponectin, and sEV microRNAs.

Indexed as

Adipose TissueDiabetes Mellitus, Type 2ObesityWeight LossAdiponectinAdultFemaleHumansInsulin ResistanceLeptinMaleMicroRNAsMiddle AgedPlasminogen Activator Inhibitor 1AdiponectinLeptinMicroRNAsPlasminogen Activator Inhibitor 1SERPINE1 protein, human

Identifiers

PMID40208704
PMCPMC12281976

What Socratic holds

Textmetadata
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Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.