ReviewInternational journal of impotence research2026
Update on the prevalence and pathophysiology of sickle cell priapism: a narrative review.
Review in International journal of impotence research, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
2 citing papers in PubMed.
- Identification of Clinical Sub-Phenotypes of Sickle Cell Disease Using Latent Class Analysis.American journal of hematology · 2026Article
- Exploring the temporal relationship between psychosocial risk factors and recurrent ischemic priapism.Sexual medicine · 2026Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Priapism is an erectile disorder with persistent and uncontrolled erections without sexual purpose. Ischemic priapism is the most frequent form of priapism and sickle cell disease is a risk factor to develop ischemic priapism with prevalence rates between 30% and 40%. The clear pathogenesis of sickle cell is still under investigation but various mechanisms including nitric oxide, phosphodiesterase type 5, opiorphin and adenosine, testosterone, RhoA/ROCK modulated by oxidative stress and genetic features are known to be key factors of this vascular disease. In this review, we explore these multiple regulatory pathways involved in the pathophysiology of priapism.
Indexed as
Identifiers
40217104What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.