Evidence mapPaperPMID 40243565Full record

ReviewInternational journal of molecular sciences2025

Metabolic-Dysfunction-Associated Steatotic Liver Disease: Molecular Mechanisms, Clinical Implications, and Emerging Therapeutic Strategies.

Jeysson E Mejía-Guzmán, Ramón A Belmont-Hernández, Norberto C Chávez-Tapia, Misael Uribe, Natalia Nuño-Lámbarri

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 46 papers.

0numbers the graph read from it
0cells of the map it votes in
46citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

46 citing papers in PubMed.

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  17. Targeted Mitochondrial ECSIT Overexpression Attenuates MASH by Increasing OTUD3 Expression.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026
    Article
  18. Current management of MASLD in type 2 diabetes.Hormones (Athens, Greece) · 2026
    Review
  19. Review
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Jeysson E Mejía-GuzmánTranslational Research Unit, Medica Sur Clinic & Foundation, Mexico City 14050, Mexico.
Ramón A Belmont-HernándezTranslational Research Unit, Medica Sur Clinic & Foundation, Mexico City 14050, Mexico.
Norberto C Chávez-TapiaTranslational Research Unit, Medica Sur Clinic & Foundation, Mexico City 14050, Mexico.ORCID 0000-0002-7451-3306
Misael UribeObesity and Digestive Diseases Unit, Medica Sur Clinic & Foundation, Mexico City 14050, Mexico.ORCID 0000-0002-6514-7869
Natalia Nuño-LámbarriTranslational Research Unit, Medica Sur Clinic & Foundation, Mexico City 14050, Mexico.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Metabolic-dysfunction-associated steatotic liver disease (MASLD), previously known as non-alcoholic fatty liver disease (NAFLD), is a highly prevalent metabolic disorder characterized by hepatic steatosis in conjunction with at least one cardiometabolic risk factor, such as obesity, type 2 diabetes, hypertension, or dyslipidemia. As global rates of obesity and metabolic syndrome continue to rise, MASLD is becoming a major public health concern, with projections indicating a substantial increase in prevalence over the coming decades. The disease spectrum ranges from simple steatosis to metabolic-dysfunction-associated steatohepatitis (MASH), fibrosis, cirrhosis, and hepatocellular carcinoma, contributing to significant morbidity and mortality worldwide. This review delves into the molecular mechanisms driving MASLD pathogenesis, including dysregulation of lipid metabolism, chronic inflammation, oxidative stress, mitochondrial dysfunction, and gut microbiota alterations. Recent advances in research have highlighted the role of genetic and epigenetic factors in disease progression, as well as novel therapeutic targets such as peroxisome proliferator-activated receptors (PPARs), fibroblast growth factors, and thyroid hormone receptor beta agonists. Given the multifaceted nature of MASLD, a multidisciplinary approach integrating early diagnosis, molecular insights, lifestyle interventions, and personalized therapies is critical. This review underscores the urgent need for continued research into innovative treatment strategies and precision medicine approaches to halt MASLD progression and improve patient outcomes.

Indexed as

Non-alcoholic Fatty Liver DiseaseAnimalsGastrointestinal MicrobiomeHumansLipid MetabolismOxidative Stressbiomarkerschronic inflammationemerging therapieslipogenesisMASLDoxidative stress

Identifiers

PMID40243565
PMCPMC11988898

What Socratic holds

Textmetadata
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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.