Evidence map›Paper›PMID 40244087›Full record

ArticleInternational journal of molecular sciences2025

Profiling Blood-Based Neural Biomarkers and Cytokines in Experimental Autoimmune Encephalomyelitis Model of Multiple Sclerosis Using Single-Molecule Array Technology.

Insha Zahoor, Sajad Mir, Shailendra Giri

Abstract read
In one paragraph

Article in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Review
  2. Review
  3. Review
  4. Blood metabolomics improves prediction of central nervous system damage in multiple sclerosis.Metabolomics : Official journal of the Metabolomic Society · 2025
    Article
  5. Article
  6. Article
  7. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

3 authors.

Insha ZahoorDepartment of Neurology, Henry Ford Health, Detroit, MI 48202, USA.ORCID 0000-0001-9919-1959
Sajad MirDepartment of Neurology, Henry Ford Health, Detroit, MI 48202, USA.
Shailendra GiriDepartment of Neurology, Henry Ford Health, Detroit, MI 48202, USA.ORCID 0000-0002-7123-829X

Funding

Novel Regulation and Targeting of Macrophages Metabolism in Neuroinflammatory DisordersR01AI144004 · NIAID · HENRY FORD HEALTH SYSTEM · PI GIRI, SHAILENDRA · 2019 to 2023
$1.9M
Endogenous metabolite restricts GM-CSF signaling pathway in pathogenic macrophages to ameliorate CNS AutoimmunityR01NS112727 · NINDS · HENRY FORD HEALTH SYSTEM · PI GIRI, SHAILENDRA · 2019 to 2023
$1.6M
Henry Ford Health Internal Support A10270 and A30967National Multiple Sclerosis Society (US) RG-1807-31964 and RG-2111-38733NIAID NIH HHS R01 AI144004NIH HHS NS112727 and AI144004NINDS NIH HHS R01 NS112727
6 · The paper itself

Abstract

Experimental autoimmune encephalomyelitis (EAE) is a preclinical animal model widely used to study multiple sclerosis (MS). Blood-based analytes, including cytokines and neural biomarkers are the predictors of neurodegeneration, disease activity, and disability in patients with MS. However, understudied confounding factors cause variation in reports on EAE across animal strains/studies, limiting the utility of these biomarkers for predicting disease activity. In this study, we investigated blood-based analyte profiles, including neural markers (NFL and GFAP) and cytokines (IL-6, IL-17, IL-12p70, IL-10, and TNF-α), in two clinically distinct EAE models: relapsing-remitting (RR)-EAE and chronic-EAE. Ultrasensitive single-molecule array technology (SIMOA, Quanterix) was used to profile the analytes in the blood plasma of mice at the acute, chronic, and progressive phases of disease. In both models, NFL was substantially increased during post-disease onset across all phases, with a pronounced increase observed in chronic-EAE. The leakage of GFAP into peripheral blood was also greater after disease onset in both EAE models, especially in the acute phase of chronic-EAE. Among all cytokines, only IL-10 had consistently lower levels in both EAE models throughout the course of disease. This study suggests NFL, GFAP, and IL-10 as potential translational predictors of disease activity in EAE, making them potential candidates as surrogate markers for the preclinical testing of therapeutic interventions in animal models of MS.

Indexed as

BiomarkersCytokinesEncephalomyelitis, Autoimmune, ExperimentalMultiple SclerosisAnimalsDisease Models, AnimalFemaleGlial Fibrillary Acidic ProteinMiceMice, Inbred C57BLBiomarkersCytokinesGlial Fibrillary Acidic Proteinbiomarkercytokinesexperimental autoimmune encephalomyelitis (EAE)glial fibrillary acidic protein (GFAP)multiple sclerosis (MS)neurofilament light chain (NFL)single molecule array (SIMOA)

Identifiers

PMID40244087
PMCPMC11989419

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.