Evidence map›Paper›PMID 40298389›Full record

ReviewCancer discovery2025

Cancer-Associated Cachexia: Bridging Clinical Findings with Mechanistic Insights in Human Studies.

Kexin Koh, Rachel Scott, Elizabeth M Cespedes Feliciano, Tobias Janowitz, Marcus D Goncalves, Eileen P White, Barry J A Laird, Kerstin Haase, Mariam Jamal-Hanjani

Abstract readReview
In one paragraph

Review in Cancer discovery, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Cancer cachexia: a caregiver and advocate perspective.Current opinion in supportive and palliative care · 2026
    Review
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  5. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Kexin KohCancer Metastasis Laboratory, University College London Cancer Institute, London, United Kingdom.ORCID 0000-0002-5783-604X
Rachel ScottCancer Metastasis Laboratory, University College London Cancer Institute, London, United Kingdom.ORCID 0009-0002-8442-7118
Elizabeth M Cespedes FelicianoDivision of Research, Kaiser Permanente Northern California, Pleasanton, California.ORCID 0000-0003-1192-4017
Tobias JanowitzCold Spring Harbor Laboratory, Cold Spring Harbor, New York, New York.ORCID 0000-0002-7820-3727
Marcus D GoncalvesDepartment of Medicine, New York University Grossman School of Medicine, New York, New York.ORCID 0000-0002-0784-9248
Eileen P WhiteRutgers Cancer Institute, New Brunswick, New Jersey.ORCID 0000-0003-2961-3065
Barry J A LairdEdinburgh Cancer Research Centre, Institute of Genetics and Cancer, University of Edinburgh, Edinburgh, United Kingdom.ORCID 0000-0002-2807-6192
Kerstin HaaseCancer Metastasis Laboratory, University College London Cancer Institute, London, United Kingdom.ORCID 0000-0002-0944-5618
Mariam Jamal-HanjaniCancer Metastasis Laboratory, University College London Cancer Institute, London, United Kingdom.ORCID 0000-0003-1212-1259

Funding

CANCAN - COLDSPRINGOT2CA278690 · NCI · COLD SPRING HARBOR LABORATORY · PI Tobias Janowitz · 2022 to 2026
$3.0M
CANcer Cachexia Action Network/CANCANOT2CA278701 · NCI · UNIVERSITY COLLEGE LONDON · PI JAMAL-HANJANI, MARIAM · 2022 to 2024
$624k
NCI NIH HHS OT2 CA278690NCI NIH HHS OT2 CA278701
6 · The paper itself

Abstract

Cancer-associated cachexia (CAC) is a chronic wasting disease typically associated with advanced cancer, resulting in progressive and debilitating loss of function and poor tolerance to anticancer therapy. Preclinical animal models have identified various potential mechanisms and mediators, which have had limited translational success in clinical trials. This review focuses on human studies and discusses the clinical phenotyping of CAC using imaging-derived body composition, quality-of-life and functional measures, existing evidence for mediators, current therapeutic options, and future directions to advance the field. Identifying mechanisms driving CAC and targeting them are expected to improve the quality of life, treatment efficacy, and survival. SIGNIFICANCE: CAC represents a significant clinical unmet need. Despite its high prevalence and associated mortality and morbidity, there are currently no globally approved effective therapies. This review provides a comprehensive overview of human studies aimed at defining CAC clinically and identifying mediators underlying it that are revealing effective health interventions. Furthermore, we highlight ongoing international efforts to advance our understanding of CAC.

Indexed as

CachexiaNeoplasmsAnimalsHumansQuality of Life

Identifiers

PMID40298389
PMCPMC12794921

What Socratic holds

Textmetadata
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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.