Evidence mapPaperPMID 40305271Full record

ReviewBiomolecules2025

The Emerging Roles of Vacuolar-Type ATPase-Dependent Lysosomal Acidification in Cardiovascular Disease.

Yan-Yan Chen, Cai-Xia Liu, Hai-Xin Liu, Shi-Yuan Wen

Abstract readReview
In one paragraph

Review in Biomolecules, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

  1. Functional genomic profiling of schizophrenia-associated genes reveals key microglial regulators.Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Yan-Yan ChenSchool of Medicine, Jiangsu University, Zhenjiang 212013, China.ORCID 0000-0002-2416-0258
Cai-Xia LiuCollege of Traditional Chinese Medicine and Food Engineering, Shanxi University of Chinese Medicine, Taiyuan 030024, China.
Hai-Xin LiuCollege of Traditional Chinese Medicine and Food Engineering, Shanxi University of Chinese Medicine, Taiyuan 030024, China.
Shi-Yuan WenCollege of Basic Medical Sciences, Shanxi Medical University, Taiyuan 030001, China.ORCID 0000-0001-9589-5676

Funding

Basic Science (Natural Science) Research Projects of Higher Education Institutions in Jiangsu Province 24KJB320001National Natural Science Foundation of China 82300531PhD Start-up Foundation of Shanxi Medical University XD2021PhD Start-up Foundation of Shanxi University of Chinese Medicine 2022BK03Shanxi Province Basic Research Program Funded Project 202203021211219
6 · The paper itself

Abstract

The vacuolar-type ATPase (V-ATPase) is a multi-subunit enzyme complex that maintains lysosomal acidification, a critical process for cellular homeostasis. By controlling the pH within lysosomes, V-ATPase contributes to overall cellular homeostasis, helping to maintain a balance between the degradation and synthesis of cellular components. Dysfunction of V-ATPase impairs lysosomal acidification, leading to the accumulation of undigested materials and contributing to various diseases, including cardiovascular diseases (CVDs) like atherosclerosis and myocardial disease. Furthermore, V-ATPase's role in lysosomal function suggests potential therapeutic strategies targeting this enzyme complex to mitigate cardiovascular disease progression. Understanding the mechanisms by which V-ATPase influences cardiovascular pathology is essential for developing novel treatments aimed at improving outcomes in patients with heart and vascular diseases.

Indexed as

Cardiovascular DiseasesLysosomesVacuolar Proton-Translocating ATPasesAnimalsHumansHydrogen-Ion ConcentrationVacuolar Proton-Translocating ATPasescardiovascular diseaselysosomal acidificationvacuolar-type ATPase

Identifiers

PMID40305271
PMCPMC12024769

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.