Evidence mapPaperPMID 40332804Full record

ReviewInternational journal of molecular sciences2025

Recent Advances in Drug Development for Alzheimer's Disease: A Comprehensive Review.

Haonan Xing, Song Yue, Runtian Qin, Xiaoxue Du, Yili Wu, Dongting Zhangsun, Sulan Luo

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 22 papers.

0numbers the graph read from it
0cells of the map it votes in
22citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

22 citing papers in PubMed.

  1. Review
  2. Article
  3. Article
  4. Review
  5. Article
  6. Article
  7. Article
  8. Neurotransmitter Systems in Alzheimer's Disease.Current issues in molecular biology · 2026
    Review
  9. Review
  10. Article
  11. Novel cinnamic acid-basedRSC advances · 2026
    Article
  12. Review
  13. Article
  14. Advances in the treatment of Alzheimer's disease.Frontiers in pharmacology · 2026
    Review
  15. Article
  16. Review
  17. Review
  18. Article
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Haonan XingGuangxi Key Laboratory of Special Biomedicine, School of Medicine, Guangxi University, Nanning 530004, China.
Song YueGuangxi Key Laboratory of Special Biomedicine, School of Medicine, Guangxi University, Nanning 530004, China.
Runtian QinGuangxi Key Laboratory of Special Biomedicine, School of Medicine, Guangxi University, Nanning 530004, China.
Xiaoxue DuGuangxi Key Laboratory of Special Biomedicine, School of Medicine, Guangxi University, Nanning 530004, China.ORCID 0009-0004-9293-0009
Yili WuKey Laboratory of Alzheimer's Disease of Zhejiang Province, Wenzhou Key Laboratory of Basic and Translational Research for Mental Disorders, Zhejiang Provincial Clinical Research Center for Mental Health, School of Mental Health, Institute of Aging, Wenzhou Medical University, Wenzhou 325000, China.
Dongting ZhangsunGuangxi Key Laboratory of Special Biomedicine, School of Medicine, Guangxi University, Nanning 530004, China.
Sulan LuoGuangxi Key Laboratory of Special Biomedicine, School of Medicine, Guangxi University, Nanning 530004, China.ORCID 0000-0002-9358-5970

Funding

"Dai Tu Yi Zhi" Project of Key R & D Program of Guangxi GUIKE AA25069003Innovation Project of Guangxi Graduate Education YCSW2024067Key Project of Natural Science Foundation of Guangxi 2024GXNSFDA999003Major Intergovernmental Joint Research Project of National Key R & D Program of China 2022YFE0132700Major International Joint Research Project of National Natural Science Foundation of China 82320108019the 111 Center D20010the National Natural Science Foundation of China 42376112the National Natural Science Foundation of China 82360698
6 · The paper itself

Abstract

Alzheimer's disease (AD) is a prevalent neurodegenerative disorder characterized by cognitive impairments such as memory loss and executive dysfunction. The primary pathological features of AD include the deposition of amyloid-beta (Aβ) plaques, the hyperphosphorylation of tau proteins leading to neurofibrillary tangles, disruptions of neuronal and synaptic functions, and chronic inflammatory responses. These multifactorial interactions drive disease progression. To date, various therapeutic agents targeting these pathological mechanisms have been developed. This article provides a comprehensive review of the pathogenesis of AD, recent advances in drug development targeting different pathways, current challenges, and future directions, aiming to offer valuable insights for clinical treatment and research.

Indexed as

Alzheimer DiseaseDrug DevelopmentAmyloid beta-PeptidesAnimalsHumanstau ProteinsAmyloid beta-Peptidestau ProteinsAlzheimer’s diseasepathogenesisreviewtherapeutics

Identifiers

PMID40332804
PMCPMC12028297

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.