Evidence mapPaperPMID 40339110Full record

ArticleThe Journal of clinical endocrinology and metabolism2025

Genomic Correlations, Shared Loci, and Drug Targets Between Polycystic Ovary Syndrome and Asthma: Insights From Genome-wide Association Analysis.

Enting Ji, Ze Wang, Ran Zhong, Yao Lu, Ruqun Zheng, Shuting Ning, Min Hu, Juan Li, Maohua Lai, Guowei Xue and 1 more

Abstract read
In one paragraph

Article in The Journal of clinical endocrinology and metabolism, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Enting JiDepartment of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, China.
Ze WangDepartment of Thoracic Surgery and Oncology, State Key Laboratory of Respiratory Disease, National Center for Respiratory Medicine, National Clinical Research Center for Respiratory Disease, Guangzhou Institute of Respiratory Health, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, China.
Ran ZhongDepartment of Thoracic Surgery and Oncology, State Key Laboratory of Respiratory Disease, National Center for Respiratory Medicine, National Clinical Research Center for Respiratory Disease, Guangzhou Institute of Respiratory Health, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, China.
Yao LuThe Sixth Clinical Medical College, Guangzhou University of Chinese Medicine, Guangzhou 510405, China.
Ruqun ZhengDepartment of Obstetrics and Gynaecology, The Chinese University of Hong Kong, Hong Kong 99907, China.
Shuting NingDepartment of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, China.
Min HuDepartment of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, China.
Juan LiDepartment of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, China.
Maohua LaiDepartment of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, China.
Guowei XueThe Sixth Clinical Medical College, Guangzhou University of Chinese Medicine, Guangzhou 510405, China.
Hongxia MaDepartment of Traditional Chinese Medicine, The First Affiliated Hospital of Guangzhou Medical University, Guangzhou 510120, China.ORCID 0000-0002-4477-613X

Funding

National Natural Science Foundation of China 82374501Natural Science Foundation of Guangdong Province 2019A1515011240
6 · The paper itself

Abstract

backgroundObservational studies have shown an association between polycystic ovary syndrome (PCOS) and asthma-related traits. However, whether this association is genetically driven or arises from observational biases remains unclear.

methodsThis study integrated data from 10 074 PCOS cases and asthma-related traits obtained from the UK Biobank and FinnGen cohorts. Global and local genetic architectures were examined using pleiotropic analysis under the composite null hypothesis, functional mapping and annotation of genetic associations, and fine-mapping credible set analysis. Drug database mining was employed to identify pleiotropic genes as potential therapeutic targets. Tissue and cell enrichment analyses were conducted to uncover shared biological mechanisms.

resultsWe identified 3 novel significant genetic loci for asthma subtypes (2 for allergic asthma and 1 for childhood asthma). A positive overall genetic correlation between PCOS and asthma-related traits was observed. We discovered 5 pleiotropic causal regions encompassing 13 genes, with ERBB3 emerging as a potential central gene contributing to the shared pathophysiology of PCOS and asthma-related traits. Additionally, drug repositioning analysis suggested anakinra and artenimol as potential therapeutic candidates for PCOS and asthma comorbidity. The linkage disequilibrium score for the specific expression of genes analysis, along with transcriptome-wide association studies, further identified gene expression patterns at the tissue/cell level in the hypothalamo-pituitary, exocrine/endocrine, respiratory, and urogenital systems.

conclusionOur findings provide novel insights into the genetic basis and biological processes underlying the association between PCOS and asthma-related traits, warranting evaluation of whether PCOS-specific asthma risk assessment could improve clinical outcomes.

Indexed as

AsthmaPolycystic Ovary SyndromeAdultDrug RepositioningFemaleGenetic Predisposition to DiseaseGenome-Wide Association StudyHumansPolymorphism, Single Nucleotideasthmagenetic architecturegenome-wide association studiespolycystic ovary syndrome

Identifiers

PMID40339110
PMCPMC12342372

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.