Evidence mapPaperPMID 40362167Full record

ReviewInternational journal of molecular sciences2025

Molecular and Pathophysiological Mechanisms Leading to Ischemic Heart Disease in Patients with Diabetes Mellitus.

Stefan Juricic, Jovana Klac, Sinisa Stojkovic, Milorad Tesic, Ivana Jovanovic, Srdjan Aleksandric, Milan Dobric, Stefan Zivkovic, Bojan Maricic, Dejan Simeunovic and 4 more

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors.

Stefan JuricicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0002-3045-5263
Jovana KlacDepartment of Cardiology, Emergency Center, University Clinical Center of Serbia, 11000 Belgrade, Serbia.
Sinisa StojkovicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0002-5784-9377
Milorad TesicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0002-3758-3719
Ivana JovanovicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.
Srdjan AleksandricClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0001-6903-4477
Milan DobricSchool of Medicine, University of Belgrade, 11000 Belgrade, Serbia.ORCID 0000-0001-7643-0451
Stefan ZivkovicDedinje Cardiovascular Institute, 11000 Belgrade, Serbia.ORCID 0000-0002-3952-4560
Bojan MaricicClinic of Cardiology, University Clinical Center Nis, 18000 Nis, Serbia.
Dejan SimeunovicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.
Ratko LasicaDepartment of Cardiology, Emergency Center, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0001-9033-5995
Miodrag DikicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.
Marko BanovicClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.
Branko BeleslinClinic for Cardiology, University Clinical Center of Serbia, 11000 Belgrade, Serbia.ORCID 0000-0001-9452-8241

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Coronary atherosclerosis in patients with diabetes mellitus is the most significant pathophysiological mechanism responsible for ischemic heart disease. Atherosclerosis in diabetes is premature, more diffuse, and more progressive, and it affects more coronary blood vessels compared to non-diabetics. Atherosclerosis begins with endothelial dysfunction, continues with the formation of fatty streaks in the intima of coronary arteries, and ends with the appearance of an atherosclerotic plaque that expands centrifugally and remodels the coronary artery. If the atherosclerotic plaque is injured, a thrombus forms at the site of the damage, which can lead to vessel occlusion and potentially fatal consequences. Diabetes mellitus and atherosclerosis are connected through several pathological pathways. Among the most significant factors that lead to atherosclerosis in diabetics are hyperglycemia, insulin resistance, oxidative stress, dyslipidemia, and chronic inflammation. Chronic inflammation is currently considered one of the most important factors in the development of atherosclerosis. However, to date, no adequate anti-inflammatory therapeutic measures have been found to prevent the progression of the atherosclerotic process, and they remain a subject of ongoing research. In this review, we summarize the most significant pathophysiological mechanisms that link atherosclerosis and diabetes mellitus.

Indexed as

Diabetes ComplicationsDiabetes MellitusMyocardial IschemiaAnimalsAtherosclerosisCoronary Artery DiseaseHumansInflammationInsulin ResistanceOxidative Stressatherosclerosischronic inflammationdiabetes mellitusdyslipidemiahyperglycemiaoxidative stress

Identifiers

PMID40362167
PMCPMC12071796

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.