Article in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
14 authors.
Mayuko HayashiAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.
Seii OhkaAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.ORCID 0000-0003-2394-6440
Daisuke NishizawaAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.ORCID 0000-0001-8286-4651
Rie InoueAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.
Masakazu HayashidaAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.ORCID 0000-0002-3598-2281
Junko HasegawaAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.
Kyoko NakayamaAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.
Yuko EbataAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.
Yuna KangAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.
Kaori YoshidaAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.
Kyotaro KoshikaDepartment of Dental Anesthesiology, Tokyo Dental College, Chiyoda-ku, Tokyo 101-0061, Japan.ORCID 0000-0002-2656-9698
Ken-Ichi FukudaDepartment of Oral Health and Clinical Science, Tokyo Dental College, Chiyoda-ku, Tokyo 101-0061, Japan.
Tatsuya IchinoheDepartment of Dental Anesthesiology, Tokyo Dental College, Chiyoda-ku, Tokyo 101-0061, Japan.ORCID 0000-0001-5793-3379
Kazutaka IkedaAddictive Substance Project, Tokyo Metropolitan Institute of Medical Science, Setagaya-ku, Tokyo 156-8506, Japan.ORCID 0000-0001-8342-0278
Funding
Japan Society for the Promotion of Science 17H04324Japan Society for the Promotion of Science 17K08970Japan Society for the Promotion of Science 19K19212Japan Society for the Promotion of Science 20K07774Japan Society for the Promotion of Science 20K09259Japan Society for the Promotion of Science 21H03028Japan Society for the Promotion of Science 22H04922Japan Society for the Promotion of Science 23K06839Japan Society for the Promotion of Science 23K21457
6 · The paper itself
Abstract
Platelet-activating factor (PAF) is a potent inflammatory mediator that activates the PAF receptor, which induces additional PAF production. Animal studies have shown that PAF induces inflammatory and neuropathic pain, including dysesthesia, a prodromal symptom of neuropathic pain. However, in humans, the association between PAF and pain remains unknown. Phospholipase A2 Group VII (PLA2G7) hydrolyzes PAF to eliminate PAF activity. The present study investigated the association between the
Indexed as
1-Alkyl-2-acetylglycerophosphocholine EsterasePolymorphism, Single NucleotidePostoperative PainAdultAgedFemaleGenetic Predisposition to DiseaseGenotypeHumansMaleMiddle Aged1-Alkyl-2-acetylglycerophosphocholine EsterasePLA2G7 protein, humandysesthesiaPAFPAF-AHPLA2G7postoperative painSNP
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.