Evidence mapPaperPMID 40427486Full record

ReviewAntioxidants (Basel, Switzerland)2025

Environmental Toxins and Oxidative Stress: The Link to Cardiovascular Diseases.

Rasheed O Sule, Gabriela Del Toro Rivera, Tanishq Vaidya, Emily Gartrell, Aldrin V Gomes

Abstract readReview
In one paragraph

Review in Antioxidants (Basel, Switzerland), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Review
  3. Article
  4. The toxic footprint of lead exposure: oxido-inflammatory biomarkers in urban populations of Niger Delta, Nigeria.Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine · 2026
    Article
  5. Review
  6. Article
  7. Article
  8. Review
  9. Article
  10. Article
  11. Review
  12. Toxicology research · 2026
    Article
  13. Article
  14. Review
  15. Article
  16. Ameliorative effects of aFrontiers in pharmacology · 2026
    Article
  17. Review
  18. Review
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Rasheed O SuleDepartment of Neurobiology, Physiology, and Behavior, University of California, Davis, CA 95616, USA.ORCID 0000-0001-9759-0809
Gabriela Del Toro RiveraDepartment of Neurobiology, Physiology, and Behavior, University of California, Davis, CA 95616, USA.
Tanishq VaidyaDepartment of Neurobiology, Physiology, and Behavior, University of California, Davis, CA 95616, USA.
Emily GartrellDepartment of Neurobiology, Physiology, and Behavior, University of California, Davis, CA 95616, USA.
Aldrin V GomesDepartment of Neurobiology, Physiology, and Behavior, University of California, Davis, CA 95616, USA.

Funding

NIH HHS 1P42ES004699-25A1
6 · The paper itself

Abstract

Cardiovascular diseases (CVDs) remain a leading global health concern, responsible for substantial morbidity and mortality. In recent years, as our understanding of the multifaceted nature of CVDs has increased, it has become increasingly evident that traditional risk factors alone do not account for the entirety of cardiovascular morbidity and mortality. Environmental toxins, a heterogeneous group of substances ubiquitous in our surroundings, have now entered the spotlight as offenders in the development and progression of CVDs. Environmental toxins include heavy metals, air pollutants, pesticides, and endocrine-disrupting chemicals, among others. Upon exposure, they can elicit oxidative stress, a condition characterized by an imbalance between the production of reactive oxygen species (ROS) and the body's ability to detoxify and repair the resulting damage. Oxidative stress triggers a cascade of events, including inflammation, endothelial dysfunction, lipid peroxidation, and vascular remodeling, which can contribute to the development of atherosclerosis, hypertension, and other cardiovascular pathologies. This article delves into the molecular mechanisms underpinning oxidative stress-mediated cardiovascular damage induced by environmental toxins, emphasizing the role of specific toxins in this process. Further research is necessary to understand how individual susceptibility and genotype influence the impact of environmental toxins on oxidative stress and the risk of CVD.

Indexed as

air pollutioncardiovascular diseasesER stressinhaled particulate matterleadmercurymicroplasticsmitochondrial dysfunctionmoldoxidative stress

Identifiers

PMID40427486
PMCPMC12108754

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.