Evidence mapPaperPMID 40453371Full record

ArticleComputational and structural biotechnology journal2025

Harnessing miRNA dynamics in HIV-1-infected macrophages: Unveiling new targeted therapeutics using systems biology.

R Harshithkumar, Mollina Kaul, Madhuri Chandane-Tak, Nikhat J Siddiqi, Abdul Malik, Abdul Arif Khan, Anupam Mukherjee

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Article in Computational and structural biotechnology journal, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

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1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

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3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

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4 · The record

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5 · Who and what money

Authors and funding

7 authors.

R HarshithkumarICMR-National Institute of Translational Virology and AIDS Research, Pune, MH, India.
Mollina KaulICMR-National Institute of Translational Virology and AIDS Research, Pune, MH, India.
Madhuri Chandane-TakICMR-National Institute of Translational Virology and AIDS Research, Pune, MH, India.
Nikhat J SiddiqiKing Saud University, Riyadh, Saudi Arabia.
Abdul MalikKing Saud University, Riyadh, Saudi Arabia.
Abdul Arif KhanICMR-National Institute of Translational Virology and AIDS Research, Pune, MH, India.
Anupam MukherjeeICMR-National Institute of Translational Virology and AIDS Research, Pune, MH, India.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: The interaction between HIV-1 and host immune cells, particularly macrophages, is crucial in understanding viral persistence and pathogenesis. This study aims to explore the impact of HIV-1 infection on macrophage microRNA (miRNA) expression profiles using a systems biology approach to uncover the potential role of miRNAs in modulating macrophage functionality and identify key miRNA targets that may serve as therapeutic avenues. Methods: PMA-differentiated THP-1 cells were used to model macrophage infection with HIV-1. A custom miRNA microarray was performed to identify dysregulated miRNAs following infection. miRTarBase was utilized for miRNA target identification, revealing gene targets associated with the dysregulated miRNAs. A protein-protein interaction (PPI) map of miRNA targets and their first interactors was constructed, with key nodes identified based on a calculated disease score, which considered degree, betweenness centrality, average shortest path length, and clustering coefficient. Gene Ontology molecular function analysis was also conducted on the identified targets. Results: The miRNA microarray identified 23 dysregulated miRNAs in HIV-1-infected macrophages, with 8 upregulated and 15 downregulated. Among these, the top 10 dysregulated miRNAs targeted over 2000 unique genes. PPI analysis revealed key nodes in the upregulated miRNA network, including Conclusions: HIV-1 infection induces significant dysregulation of miRNAs in macrophages, impacting a wide array of gene targets and molecular functions. These findings suggest that miRNA-mediated regulation may play a crucial role in HIV-1 pathogenesis within macrophages and present potential targets for miRNA-based therapeutic strategies.

Indexed as

Biological networksGene ontologyHIV-1MicroarrayMiRNAMolecular functionPathogenesis

Identifiers

PMID40453371
PMCPMC12124685

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.